Although the genetic contribution to fragile X syndrome (FXS) is well understood, associated neurological impairments resulting from this mutation, and their cognitive sequelae, continue to be explored. These efforts are undoubtedly enhanced by remarkable developments in neuroscience measurement tools, as demonstrated by Haas et al. in their diffusion tensor imaging (DTI) study of FXS in this issue.1 DTI is a relatively recent structural imaging technique that allows an investigation of the integrity of white matter fibres along specific neuronal pathways.
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