首页> 外文期刊>The Journal of foot and ankle surgery: official publication of the American College of Foot and Ankle Surgeons >Surgical Reconstruction and Mobilization Therapy for a Retracted Extensor Hallucis Longus Laceration and Tendon Defect Repaired by Split Extensor Hallucis Longus Tendon Lengthening and Dermal Scaffold Augmentation
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Surgical Reconstruction and Mobilization Therapy for a Retracted Extensor Hallucis Longus Laceration and Tendon Defect Repaired by Split Extensor Hallucis Longus Tendon Lengthening and Dermal Scaffold Augmentation

机译:分离式伸指拇长肌腱加长和真皮支架隆起修复伸肌伸肌撕裂和肌腱缺损的手术重建和动员治疗

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摘要

A reconstructive technique and physical therapy protocol is presented for the treatment of extensor hallucis longus (EHL) lacerations with critical size defects caused by tendon retraction. The primary goal of treatment was to restore EHL structure and function without the use of a bridging allograft or tendon transfer. The technique is performed by split lengthening the distal segment of the lacerated EHL and rotating the lengthened segment proximally 180° to bridge the tendon defect. The lengthened tendon is then sutured to the proximal segment of the EHL. The EHL is then tubularized with an acellular dermal scaffold at the region of tendon rotation to improve tendon strength, minimize the probability of tendon overlengthening or re-rupture, and improve the tendon gliding motion, which can be compromised by the tendon irregularity caused by rotation of the tendon. Postoperative range of motion therapy should be initiated at 3 weeks postoperatively. A case report of this technique and postoperative mobilization protocol is presented. The American Orthopaedic Foot and Ankle Society midfoot score at 3 and 6 months postoperatively was 90 of 100. The patient regained active dorsiflexion motion of the hallux without functional limitations, deformity, or contracture of the hallux. The advantages of this technique include that a large cadaveric allograft is not needed to bridge a critical size tendon defect and tendon lengthening provides a biologically active tendon graft without the secondary comorbidities and dysfunction commonly associated with tendon transfer procedures.
机译:提出了一种重建技术和物理疗法方案,用于治疗由伸肌引起的临界尺寸缺陷的长伸肌(EHL)撕裂伤。治疗的主要目的是在不使用桥接同种异体移植或腱移植的情况下恢复EHL的结构和功能。通过劈开加长的EHL远端段并向近端旋转加长的段180°以桥接肌腱缺损来执行该技术。然后将加长的肌腱缝合到EHL的近端节段。然后在腱旋转区域用无细胞真皮支架对EHL进行管状化,以提高腱强度,最小化腱过度伸长或再次断裂的可能性,并改善腱滑动运动,这可能会因旋转引起的腱不规则而受损肌腱。术后3周应开始进行运动治疗。提出了该技术和术后动员方案的病例报告。术后3个月和6个月,美国骨科足踝学会中足评分为90分(满分100分)。患者恢复了拇趾的主动背屈运动,没有功能受限,畸形或挛缩。该技术的优点包括不需要大的尸体同种异体桥来弥合临界尺寸的肌腱缺损,并且肌腱延长提供了具有生物学活性的肌腱移植物,而没有通常与肌腱转移程序相关的继发合并症和功能障碍。

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