机译:细胞因子受体信号传导的拓扑控制诱导造血作用的差异
Stanford Univ, Sch Med, Dept Mol & Cellular Physiol, Stanford, CA 94305 USA;
Univ Washington, Dept Biochem, Seattle, WA 98195 USA|Univ Washington, Inst Prot Design, Seattle, WA 98195 USA;
Harvard Med Sch, Boston Childrens Hosp, Manton Ctr Orphan Dis Res, Div Hematol Oncol, Boston, MA 02115 USA|Harvard Med Sch, Dana Farber Canc Inst, Dept Pediat Oncol, Boston, MA 02115 USA|Broad Inst MIT & Harvard, Cambridge, MA 02142 USA;
Stanford Univ, Howard Hughes Med Inst, Sch Med, Stanford, CA 94305 USA;
Univ Washington, Dept Biochem, Seattle, WA 98195 USA|Univ Washington, Inst Prot Design, Seattle, WA 98195 USA;
Nankai Univ, State Key Lab Med Chem Biol, Tianjin, Peoples R China|Nankai Univ, Coll Pharm, Tianjin, Peoples R China;
Univ Osnabruck, Div Biophys, Dept Biol, D-49076 Osnabruck, Germany;
Stanford Univ, Sch Med, Dept Mol & Cellular Physiol, Stanford, CA 94305 USA;
Stanford Univ, Sch Med, Dept Mol & Cellular Physiol, Stanford, CA 94305 USA|Stanford Univ, Howard Hughes Med Inst, Sch Med, Stanford, CA 94305 USA;
Univ Osnabruck, Div Biophys, Dept Biol, D-49076 Osnabruck, Germany|Univ Osnabruck, Ctr Cellular Nanoanalyt, D-49076 Osnabruck, Germany;
Harvard Med Sch, Boston Childrens Hosp, Manton Ctr Orphan Dis Res, Div Hematol Oncol, Boston, MA 02115 USA|Harvard Med Sch, Dana Farber Canc Inst, Dept Pediat Oncol, Boston, MA 02115 USA|Broad Inst MIT & Harvard, Cambridge, MA 02142 USA;
Univ Washington, Dept Biochem, Seattle, WA 98195 USA|Univ Washington, Inst Prot Design, Seattle, WA 98195 USA|Univ Washington, Howard Hughes Med Inst, Seattle, WA 98195 USA;
Stanford Univ, Sch Med, Dept Mol & Cellular Physiol, Stanford, CA 94305 USA|Stanford Univ, Howard Hughes Med Inst, Sch Med, Stanford, CA 94305 USA|Stanford Univ, Sch Med, Dept Struct Biol, Stanford, CA 94305 USA;
机译:通过gp130 /白血病抑制因子受体复合物介导的白介素6家族细胞因子信号传导在脂质筏中的差异刺激诱导受体定位。
机译:通过造血干细胞和祖细胞将危险信号转化为细胞因子信号,用于调节应激造血的调节
机译:由I型和II型干扰素(IFN)诱导的Stat介导的信号传导通过脂质微结构域缔合和网格蛋白依赖的IFN受体胞吞作用而受到不同控制
机译:吸附的内毒素介导对细胞因子和趋化因子释放的颗粒诱导的刺激差异影响
机译:饮食诱导的肥胖症会阻止中央瘦素基因疗法的有效厌食症和脂肪减少症:肥胖症中瘦素受体表达/信号转导受损的证据以及受热量限制的逆转。
机译:由I型和II型干扰素(IFN)诱导的Stat介导的信号通过脂质微结构域关联和网格蛋白依赖的IFN受体胞吞作用而受到不同控制。
机译:教师意见拓扑控制拓扑控制的推荐诱导血液缺陷中的差异效应。