机译:p16(Ink4a)和p19(Arf)-p53通路均能抑制小鼠胰腺腺癌的进展
Harvard Univ, Sch Med, Dana Farber Canc Inst, Dept Med Oncol, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Dana Farber Canc Inst, Ctr Appl Canc Sci, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Brigham & Womens Hosp, Dept Med, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Brigham & Womens Hosp, Dept Pathol, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Dept Genet, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Dept Dermatol, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Dept Biol Chem, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Dept Mol Pharmacol, Boston, MA 02115 USA;
Harvard Univ, Sch Med, Massachusetts Gen Hosp, Ctr Canc, Boston, MA 02114 USA;
Harvard Univ, Sch Med, Massachusetts Gen Hosp, Ctr Mol Imaging Res, Boston, MA 02114 USA;
array comparative genomic hybridization; mouse models; pancreatic cancer; KRAS; tumor suppressor; TUMOR-SUPPRESSOR GENE; INTRAEPITHELIAL NEOPLASIA; DUCTAL ADENOCARCINOMA; CELL-PROLIFERATION; CANCER STATISTICS; RAS MUTATIONS; K-RAS; P53; CARCINOMA; EXPRESSION;
机译:小鼠肌原细胞的永生化不会丢失p16 INK4a,p19 ARF或p53,并且会因Bax失活而加速
机译:尽管表达了p16(Ink4a),p19(Arf),p53和p21(Waf1 / Cip1),但原代培养的小鼠肝肿瘤细胞对细胞衰老具有抗性。
机译:p16(INK4a)/ p14(ARF)和p53通路在粘液样/圆形细胞脂肪肉瘤的圆形细胞成分中频繁改变:p53基因改变和p14(ARF)表达降低均与不良预后相关。
机译:从Lincrna-ROR / miR-145 / ARF6表达途径产生三维曲线和siRNA mRNA ARF6的二维曲目,参与三重阴性乳腺癌进展
机译:ST6GAL-I介导的唾液酸化促进胰腺导管腺癌进展和化学性
机译:从封面:p16Ink4a和p19Arf-p53途径都限制了小鼠胰腺腺癌的进展
机译:p16Ink4a和p19Arf-p53途径均能抑制小鼠胰腺腺癌的进展
机译:关于遗传修饰的单倍体p16(Ink4a)/ p19(arf)小鼠中苯酚酞(Cas NO.77-09-8)的毒理学和癌变研究的NTp报告(饲料研究)。