机译:通过调节cIAP-2表达来克服癌细胞对Smac模拟物诱导的凋亡的抵抗力
Howard Hughes Medical Institute and Department of Biochemistry University of Texas Southwestern Medical Center, Dallas, TX 75390;
rnHamon Center for Therapeutic Oncology Research and Simmons Cancer Center, University of Texas Southwestern Medical Center, Dallas, TX 75390;
rnHamon Center for Therapeutic Oncology Research and Simmons Cancer Center, University of Texas Southwestern Medical Center, Dallas, TX 75390;
rnHoward Hughes Medical Institute and Department of Biochemistry University of Texas Southwestern Medical Center, Dallas, TX 75390;
TNFα; NF-κB; PI3K; Caspase-8; PRIK1;
机译:Smac模拟剂和去甲基化剂协同引发急性髓性白血病细胞死亡,并通过诱导坏死病克服细胞凋亡抗性
机译:SMAC模拟物通过引发肿瘤坏死因子α-诱导的粪便造成的粪便溃疡绕过FADD-或Caspase-8缺陷细胞中的凋亡抗性
机译:在临床前ALL异种移植体内模型中通过SMAC模拟物克服高危急性淋巴细胞白血病的细胞凋亡抗性
机译:Smac基因的过量表达增强了宫颈癌HeLa细胞辐射诱导的凋亡作用
机译:Smad3通过控制人类乳腺癌细胞中survivin的表达介导TGF-β诱导的细胞凋亡。
机译:通过调节cIAP-2表达来克服癌细胞对Smac模拟物诱导的凋亡的抵抗力
机译:通过调节cIAP-2表达来克服癌细胞对Smac模拟物诱导的凋亡的抵抗力