机译:感觉神经元中PIK3C3 / Vps34的缺失通过破坏内体而不是自噬途径而引起快速的神经变性
Department of Cell Biology, Duke University Medical Center, Durham, NC 27710 Department of Neurobiology, Duke University Medical Center, Durham, NC 27710;
rnDepartment of Cell Biology, Duke University Medical Center, Durham, NC 27710;
rnDepartment of Cell Biology, Duke University Medical Center, Durham, NC 27710;
rnDepartment of Cell Biology, Duke University Medical Center, Durham, NC 27710;
rnDepartment of Cell Biology, Duke University Medical Center, Durham, NC 27710;
rnDepartment of Orthopaedic Surgery, Keio University School of Medicine, 160-8582 Tokyo, Japan Department of Orthopaedic Surgery, National Hospital Organization, Murayama Medical Center, 208-0011 Tokyo, Japan;
rnDepartment of Immunology, Duke University Medical Center, Durham, NC 27710;
rnDepartment of Cell Biology, Duke University Medical Center, Durham, NC 27710 Department of Neurobiology, Duke University Medical Center, Durham, NC 27710;
机译:锥体神经元中Pik3c3的缺失导致突触丧失,广泛的胶质增生和进行性神经变性
机译:ATG38和NRBF2的表征,自噬VPS34 / PIK3C3复合物的第五个亚基
机译:哺乳动物PIK3C3 / VPS34:肝脏和心脏自噬过程的关键
机译:快速适应的感觉神经元动作电位编码的非线性模型
机译:雌激素受体和初级感觉神经元伤害感受性信号通路的作用。
机译:感觉神经元中PIK3C3 / Vps34的缺失通过破坏内体而不是自噬途径而引起快速的神经变性
机译:感觉神经元中PIK3C3 / Vps34的缺失通过破坏内体而不是自噬途径而引起快速的神经变性
机译:青蛙和大鼠感觉神经元中快速转运轴突蛋白的比较分析