机译:生长素基因敲除小鼠突触的内吞作用和网格蛋白脱膜缺陷
Laboratory of Cell Biology, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD 20892-0301;
Synaptic Transmission Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892-0301;
Synaptic Transmission Section, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD 20892-0301;
Department of Cell Biology and Howard Hughes Medical Institute, Program in Cellular Neuroscience, Neurodegeneration, and Repair, Yale University School of Medicine, New Haven, CT 06510;
Department of Cell Biology and Howard Hughes Medical Institute, Program in Cellular Neuroscience, Neurodegeneration, and Repair, Yale University School of Medicine, New Haven, CT 06510;
Laboratory of Cell Biology, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD 20892-0301;
Laboratory of Cell Biology, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD 20892-0301;
Hsc70; gene targeting; synaptic transmission; vesicle recycling;
机译:将Bbs10完全敲除表型与特定的肾脏上皮敲除表型进行比较,突出了小鼠肾脏缺陷与全身失活之间的联系
机译:氯离子通道和内吞作用:来自登特氏病和CIC-5基因敲除小鼠的新见解
机译:CLDN14敲除小鼠中的内毛细胞丝带突触和听觉神经纤维回归的丧失
机译:褪黑素减少了野生型小鼠中由光血栓形成引起的脑梗死的体积,而不是环氧化酶-1基因敲除小鼠
机译:新型Col5a3基因敲除和Tll1条件基因敲除小鼠的表征。
机译:生长素基因敲除小鼠突触的内吞作用和网格蛋白脱膜缺陷
机译:生长素基因敲除小鼠突触的内吞作用和网格蛋白脱膜缺陷