机译:乳酸脱氢酶A的抑制诱导氧化应激并抑制肿瘤进展
Division of Hematology, Department of Medicine,The Johns Hopkins University School of Medicine, Baltimore, MD 21205;
rnDivision of Hematology, Department of Medicine,The Johns Hopkins University School of Medicine, Baltimore, MD 21205;
rnDepartments of Pathology,The Johns Hopkins University School of Medicine, Baltimore, MD 21205;
rnDivision of Hematology, Department of Medicine,The Johns Hopkins University School of Medicine, Baltimore, MD 21205;
rnDepartments of Pathology,The Johns Hopkins University School of Medicine, Baltimore, MD 21205 Departments of Oncology, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;
rnDepartment of Chemistry, University of New Mexico, Albuquerque, NM 87131;
rnDepartment of Biochemistry and Molecular Biology, University of New Mexico School of Medicine, Albuquerque, NM 87131;
rnDepartment of Biochemistry and Molecular Biology, University of New Mexico School of Medicine, Albuquerque, NM 87131;
rnDepartments of Oncology, The Johns Hopkins University School of Medicine, Baltimore, MD 21205 Institute for Cell Engineering,The Johns Hopkins University School of Medicine, Baltimore, MD 21205 McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205 Departments of Pediatrics, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;
rnDivision of Hematology, Department of Medicine,The Johns Hopkins University School of Medicine, Baltimore, MD 21205 Departments of Pathology,The Johns Hopkins University School of Medicine, Baltimore, MD 21205 Departments of Oncology, The Johns Hopkins University School of Medicine, Baltimore, MD 21205 Departments of Molecular Biology, The Johns Hopkins University School of Medicine, Baltimore, MD 21205 Departments of Genetics, The Johns Hopkins University School of Medicine, Baltimore, MD 21205 Departments of Cell Biology,The Johns Hopkins University School of Medicine, Baltimore, MD 21205;
glycolysis; lymphoma; pancreatic cancer; redox stress; xenograft models;
机译:靶向乳酸脱氢酶-A抑制肺癌小鼠模型的肿瘤发生和肿瘤进展,并影响肿瘤起始细胞
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机译:热应激诱导eEudurilus eugeniae(Kinberg)组织蛋白,脂质过氧化和乳酸脱氢酶,乙酰胆碱酯酶和过氧化氢酶活性的变化
机译:内质网应激诱导Jpk的表达,从而抑制F9畸胎癌细胞的细胞周期进程
机译:VEGF自分泌信号在缺氧和氧化应激驱动的“茎干转换”中的作用:对实体瘤进展和转移的影响。
机译:乳酸脱氢酶A的抑制诱导氧化应激并抑制肿瘤进展
机译:乳酸脱氢酶A的抑制诱导氧化应激并抑制肿瘤进展