机译:CDX1通过诱导与干性相关的重编程因子SALL4和KLF5赋予胃上皮细胞肠道表型
Division of Microbiology, Graduate School of Medicine, The University of Tokyo, Tokyo 113-0033, Japan,Division of Chemistry, Graduate School of Science, Hokkaido University, Sapporo 060-0810, Japan;
Division of Microbiology, Graduate School of Medicine, The University of Tokyo, Tokyo 113-0033, Japan;
Division of Gastroenterology and Hepatology, Department of Internal Medicine, Keio University School of Medicine, Tokyo 160-8582, Japan;
Genome Science Division, Research Center for Advanced Science and Technology, The University of Tokyo, Tokyo 153-8904, Japan;
Division of Gastroenterology, Department of Medicine, Jichi Medical University, Tochigi 329-0498, Japan;
Department of Gastroenterology, Yokohama City University Graduate School of Medicine, Yokohama 236-0004, Japan;
Department of Gastrointestinal Surgery, Graduate School of Medicine, The University of Tokyo, Tokyo 113-8655, Japan;
Department of Gastrointestinal Surgery, Graduate School of Medicine, The University of Tokyo, Tokyo 113-8655, Japan;
Genome Science Division, Research Center for Advanced Science and Technology, The University of Tokyo, Tokyo 153-8904, Japan;
Division of Microbiology, Graduate School of Medicine, The University of Tokyo, Tokyo 113-0033, Japan;
机译:肠特异性转录因子Cdx1和Cdx2的表达与胃癌细胞中肠表型的转变相关。
机译:肠特异性转录因子Cdx1和Cdx2的表达与胃癌细胞中肠表型的转变相关
机译:脱氧胆酸(DCA)通过诱导与干性相关的重编程因子OCT4和SOX2在食道鳞状上皮细胞上赋予肠道表型
机译:体外分析人肠上皮细胞和巨噬细胞样细胞之间的相互作用
机译:通过IL-13Ralpha2诱导的MAP激酶激活和活性氧的诱导,肠上皮细胞中白介素13信号的调节。
机译:从封面:CDX1通过诱导与干性相关的重编程因子SALL4和KLF5赋予胃上皮细胞肠道表型
机译:肠上皮细胞分化标志物肠碱性磷酸酶(ALPI)被组蛋白脱乙酰酶抑制剂(HDACI)在结肠癌细胞中选择性地诱导KRUPPEL样系数5(KLF5)依赖性方式。