机译:规范的Wnt抑制剂Axin2促进结肠癌致癌活性
Division of Molecular Medicine and Genetics, Department of Internal Medicine, and the Life Sciences Institute, University of Michigan, Ann Arbor, Ml 48109;
Department of Visceral Surgery and Comprehensive Cancer Center Freiburg, University of Freiburg, 79106 Freiburg, Germany;
Division of Molecular Medicine and Genetics, Department of Internal Medicine, and the Life Sciences Institute, University of Michigan, Ann Arbor, Ml 48109;
Division of Molecular Medicine and Genetics, Department of Internal Medicine, and the Life Sciences Institute, University of Michigan, Ann Arbor, Ml 48109;
Division of Molecular Medicine and Genetics, Department of Internal Medicine, and the Life Sciences Institute, University of Michigan, Ann Arbor, Ml 48109;
Division of Molecular Medicine and Genetics, Department of Internal Medicine, and the Life Sciences Institute, University of Michigan, Ann Arbor, Ml 48109;
Division of Molecular Medicine and Genetics, Department of Internal Medicine, and the Life Sciences Institute, University of Michigan, Ann Arbor, Ml 48109;
invasion; e-cadherin; basement membrane; gsk3p; tankyrase;
机译:MiR-183通过靶向AXIN2维持经典Wnt信号传导活性并调节膀胱癌的生长和凋亡
机译:GSK-3β在调节Cononical Wnt /β-catenin信号传导和结肠癌中的PI3-K / akt致癌途径的作用
机译:非经典WNT5A信号上调肿瘤抑制因子15-PGDH的表达并诱导结肠癌细胞的分化
机译:DKK1抑制经典的Wnt信号通路诱导体外hMSC中增加的ALP活性
机译:配体-受体相互作用决定了典型Wnt通路的信号传递潜能和致癌性。
机译:规范的Wnt抑制剂Axin2促进结肠癌致癌活性
机译:HCV核心蛋白增强规范的Wnt /β-Catenin信号转导活性可促进肝癌细胞的生长
机译:人前列腺癌中人aXIN2及其他Wnt信号通路基因的分析