High glucose-induced hypertrophy of mesangial cells is reversed by connexin43 overexpression via PTEN/Akt/mTOR signaling

Xiang-Mei Chen

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High glucose-induced hypertrophy of mesangial cells is reversed by connexin43 overexpression via PTEN/Akt/mTOR signaling

机译：连接蛋白43通过PTEN / Akt / mTOR信号转导过度表达，可逆转高葡萄糖诱导的系膜细胞肥大

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《Nephrology Dialysis Transplantation》 |2012年第1期|p.90-100|共11页
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Xiang-Mei Chen;
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1. High glucose-induced hypertrophy of mesangial cells is reversed by connexin43 overexpression via PTEN/Akt/mTOR signaling [J] . LiuL., HuX., CaiG.-Y., Nephrology, dialysis, transplantation: official publication of the European Dialysis and Transplant Association - European Renal Association . 2012,第1期

机译：连接蛋白43的过表达通过PTEN / Akt / mTOR信号转导逆转了高葡萄糖诱导的系膜细胞肥大
2. Hydrophobic motif site-phosphorylated protein kinase CbetaII between mTORC2 and Akt regulates high glucose-induced mesangial cell hypertrophy [J] . Falguni Das, Nini Ghosh-Choudhury, Meenalakshmi M. Mariappan, American Journal of Physiology . 2016,第4aPta1期

机译：MTORC2和AKT之间的疏水基序位点 - 磷酸化蛋白激酶CBetaii调节高葡萄糖诱导的乳房细胞肥大
3. Cardiac overexpression of insulin-like growth factor 1 attenuates chronic alcohol intake-induced myocardial contractile dysfunction but not hypertrophy: Roles of Akt, mTOR, GSK3beta, and PTEN. [J] . Zhang B, Turdi S, Li Q, Free Radical Biology and Medicine: The Official Journal of the Oxygen Society . 2010,第7期

机译：心脏过度表达胰岛素样生长因子1可减轻慢性酒精摄入引起的心肌收缩功能障碍，但不能减轻肥大：Akt，mTOR，GSK3beta和PTEN的作用。
4. Cationic polystyrene nanosphere induces autophagy through inhibition of the Akt/mTOR and activation of the AMPK signaling pathways in macrophage and epithelial cells [C] . Hui-Wen Chiu, Tian Xia, Jui-Chen Tsai, Annual NSTI nanotechnology conference and expo . 2013

机译：阳离子聚苯乙烯纳米球通过抑制Akt / mTOR和激活巨噬细胞和上皮细胞中的AMPK信号通路来诱导自噬
5. Mechanisms of high glucose-induced altered endothelin-1 signalling in glomerular mesangial cells. [D] . Hua, Hong Nhouc. 2003

机译：高糖诱导的肾小球系膜细胞中内皮素-1信号改变的机制。
6. TGFβ-Stimulated MicroRNA-21 Utilizes PTEN to Orchestrate AKT/mTORC1 Signaling for Mesangial Cell Hypertrophy and Matrix Expansion [O] . Nirmalya Dey, Nandini Ghosh-Choudhury, Balakuntalam S. Kasinath, -1

机译：TGFβ刺激的微小RNa-21利用pTEN编排aKT / mTORC1信号对肾小球系膜细胞的肥大和基质扩张
7. TGFβ-stimulated microRNA-21 utilizes PTEN to orchestrate AKT/mTORC1 signaling for mesangial cell hypertrophy and matrix expansion. [O] . Nirmalya Dey, Nandini Ghosh-Choudhury, Balakuntalam S Kasinath, 2012

机译：TGFβ刺激的microRNa-21利用pTEN协调aKT / mTORC1信号传导，用于系膜细胞肥大和基质扩增。

High glucose-induced hypertrophy of mesangial cells is reversed by connexin43 overexpression via PTEN/Akt/mTOR signaling

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