机译:T细胞通过抑制NLRP1和NLRP3炎症小体来抑制先天性免疫反应
Department of Biochemistry, University of Lausanne, Chemin des Boveresses 155, CH-1066 Epaiinges, Switzerland;
Department of Biochemistry, University of Lausanne, Chemin des Boveresses 155, CH-1066 Epaiinges, Switzerland;
Department of Biochemistry, University of Lausanne, Chemin des Boveresses 155, CH-1066 Epaiinges, Switzerland;
Department of Biochemistry, University of Lausanne, Chemin des Boveresses 155, CH-1066 Epaiinges, Switzerland;
Department of Biochemistry, University of Lausanne, Chemin des Boveresses 155, CH-1066 Epaiinges, Switzerland;
Department of Biochemistry, University of Lausanne, Chemin des Boveresses 155, CH-1066 Epaiinges, Switzerland;
Department of Biochemistry, University of Lausanne, Chemin des Boveresses 155, CH-1066 Epaiinges, Switzerland;
Department of Biochemistry, University of Lausanne, Chemin des Boveresses 155, CH-1066 Epaiinges, Switzerland;
机译:NLRP1炎性小体的治疗中和作用降低了先天性免疫反应并改善了脑外伤后的组织病理学。
机译:出版商矫正:利什曼菊属植物RNA病毒通过通过TLR3介导的NLRP3炎症抑制来颠覆先天免疫性加剧Leishmaniaisis
机译:Leishmania RNA病毒通过通过TLR3介导的NLRP3炎症抑制来颠覆先天免疫性加剧Leishmaniaisis
机译:HCV病毒抑制用慢性丙型肝炎天然干扰素-β的诱导方法恢复先天和适应性免疫应答
机译:嗜肺军团菌的II型分泌系统通过MyD88 / TLR2途径抑制宿主的先天免疫反应。
机译:THE NLRp1炎性体THERapEUTIC中和降低了先天免疫应答并提高组织病理脑外伤后
机译:Leishmania RNA病毒通过通过TLR3介导的NLRP3炎症抑制来颠覆先天免疫性加剧Leishmaniaisis