机译:复合囊泡融合增加了量子大小并增强了突触传递
National Institute of Neurological Disorders and Stroke, 35 Convent Drive, Building 35, Room 2B-1012, Bethesda, Maryland 20892, USA;
National Institute of Neurological Disorders and Stroke, 35 Convent Drive, Building 35, Room 2B-1012, Bethesda, Maryland 20892, USA;
National Institute of Neurological Disorders and Stroke, 35 Convent Drive, Building 35, Room 2B-1012, Bethesda, Maryland 20892, USA;
National Institute of Neurological Disorders and Stroke, 35 Convent Drive, Building 35, Room 2B-1012, Bethesda, Maryland 20892, USA;
National Institute of Neurological Disorders and Stroke, 35 Convent Drive, Building 35, Room 2B-1012, Bethesda, Maryland 20892, USA;
Department of Pulmonary Medicine, The University of Texas M. D. Anderson Cancer Center, 2121 West Holcombe Boulevard, Box 1100, Houston, Texas 77030, USA;
Department of Pulmonary Medicine, The University of Texas M. D. Anderson Cancer Center, 2121 West Holcombe Boulevard, Box 1100, Houston, Texas 77030, USA;
National Institute of Neurological Disorders and Stroke, 35 Convent Drive, Building 35, Room 2B-1012, Bethesda, Maryland 20892, USA;
机译:SNARE力可以使突触小泡融合并控制定量突触传递的动力学。
机译:突触糖蛋白I缺乏症小鼠的突触小囊簇和突触传递的损伤,以及易感性
机译:突触小泡转运蛋白表达调节小泡表型和数量大小。
机译:增加突触囊泡的可释放池削弱便利
机译:活性区材料大分子的物理变化,可调节突触小泡膜与突触前膜的对接和融合以及从活性区去除融合的小泡膜。
机译:钙/突触结合蛋白诱发的化合物融合增加量子尺寸和突触强度
机译:在突触素I缺陷型小鼠中,突触小泡聚集和突触传递受损,癫痫发作倾向增加。