机译:白介素受体激活MYD88-ARNO-ARF6级联反应,破坏血管稳定性
Department of Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA;
Department of Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Department of Oncological Sciences, University of Utah, Salt Lake City, Utah 84112, USA;
Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Department of Bioengineering, University of Utah, Salt Lake City, Utah 84112, USA;
Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Department of Human Genetics, University of Utah, Salt Lake City, Utah 84112, USA;
Navigen Inc, Salt Lake City, Utah 84112, USA;
Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA;
Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Department of Human Genetics, University of Utah, Salt Lake City, Utah 84112, USA;
Department of Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Department of Anatomy, Second Military Medical University, Shanghai 200433, China;
Department of Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Department of Oncological Sciences, University of Utah, Salt Lake City, Utah 84112, USA;
Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Department of Pathology, University of Utah, Salt Lake City, Utah 84112, USA;
Department of Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA;
Department of Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Program in Molecular Medicine, University of Utah, Salt Lake City, Utah 84112, USA,Department of Oncological Sciences, University of Utah, Salt Lake City, Utah 84112, USA,Cardiology Section, VA Salt Lake City Health Care System, Salt Lake City, Utah 84112, USA,The Key Laboratory for Human Disease Gene Study of Sichuan Province, Institute of Laboratory Medicine, Sichuan Academy of Medical Sciences & Sichuan Provincial People's Hospital, Chengdu, Sichuan 610072, China;
机译:经典的白细胞介素6受体信号传导和白细胞介素6反信号差异控制血管紧张素II依赖性高血压,心脏信号转导子和转录3激活激活剂以及体内血管肥大。
机译:经典的白细胞介素6受体信号传导和白细胞介素6反信号可差异性控制血管紧张素II依赖性高血压,心脏信号转导子和转录3激活激活子以及体内血管肥大。
机译:过氧化物酶体增殖物激活受体(PPAR)在大鼠血管平滑肌细胞中抑制白介素1β诱导的IIA组分泌型磷脂酶A2表达:PPARβ与原癌基因BCL-6之间的合作
机译:为白细胞介素及其受体构建鲁棒和基于细胞的基于细胞的测定
机译:血栓烷受体激活调节血管平滑肌细胞中白介素-1β诱导的单核细胞粘附和核因子κB依赖性基因表达的信号传导机制
机译:白介素受体激活mYD88-aRNO-aRF6级联破坏血管稳定性
机译:白细胞介素受体激活MYD88-ARNO-ARF6级联以破坏血管稳定性