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机译:线粒体缺陷通过果蝇中的河马信号驱动非自主性肿瘤进展
Division of Genetics, Kobe University Graduate School of Medicine, 7-5-1 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan;
Division of Genetics, Kobe University Graduate School of Medicine, 7-5-1 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan;
Division of Genetics, Kobe University Graduate School of Medicine, 7-5-1 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan;
Division of Genetics, Kobe University Graduate School of Medicine, 7-5-1 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan;
Division of Genetics, Kobe University Graduate School of Medicine, 7-5-1 Kusunoki-cho, Chuo-ku, Kobe 650-0017, Japan;
PREST0, Japan Science and Technology Agency (JST), 4-1-8 Honcho Kawaguchi, Saitama 332-0012, Japan;
机译:河马在肿瘤进展中的河马信号和miRNA之间的串扰
机译:Rab5的丢失通过果蝇中的TNF和Ras信号传导驱动非自主细胞增殖
机译:Rab5的丢失通过果蝇中的TNF和Ras信号传导驱动非自主细胞增殖
机译:线粒体超氧化物歧化酶(SOD2)RNA干扰敲低对Drosophila melanogaster线粒体蛋白羰基化的影响
机译:线粒体产生的进展信号在晚期癌症发展中的“重新唤醒”
机译:维持成人成年果蝇的飞行肌肉功能和线粒体完整性需要通过肿瘤抑制因子PTEN对PI3K / AKT信号进行微调。
机译:果蝇中Rab5的丢失通过TNF和Ras信号传导驱动非自主细胞增殖