机译:紊乱的肽段的立体特异性结合介导BK通道失活。
Department of Anesthesiology, Washington University School of Medicine, St Louis, Missouri 63110, USA;
Department of Anesthesiology, Washington University School of Medicine, St Louis, Missouri 63110, USA,Institute of Life Science, Nanchang University, 999 Xuefu Road, Honggu District, Nanchang 330031, China.;
Department of Biochemistry, Washington University School of Medicine, St Louis, Missouri 63110, USA;
Department of Anesthesiology, Washington University School of Medicine, St Louis, Missouri 63110, USA;
机译:β2辅助亚基的NH2末端使BK通道失活:三种疏水残基的末端肽段的基本作用。
机译:β亚基的N末端失活域通过BK通道前壁中的结合保护免受胰蛋白酶消化。
机译:Rotundifolone诱导的放松是由BK(Ca)通道激活和Ca(v)通道失活介导的。
机译:门控电荷对介导平滑肌细胞BK通道双重激活的影响:计算研究。
机译:多巴胺介导的人冠状动脉平滑肌细胞BK通道活化的机制
机译:无序肽段的立体特异性结合介导BK信道灭活
机译:错误:无序肽段的立体特异性结合介导BK信道灭活