机译:HELQ促进RAD51旁系同源蛋白修复,从而避免生殖细胞丢失和肿瘤发生
DNA Damage Response Laboratory, London Research Institute, Cancer Research UK, Clare Hall, South Mimms EN6 3LD, UK;
DNA Damage Response Laboratory, London Research Institute, Cancer Research UK, Clare Hall, South Mimms EN6 3LD, UK;
Centerfor Biological Sequence Analysis. Technical University of Denmark, Lyngby 2800, Denmark;
Institute of Molecular Cancer Research, University of Zurich, Winterthurerstrasse 190, CH-8057 Zurich, Switzerland;
DNA Damage Response Laboratory, London Research Institute, Cancer Research UK, Clare Hall, South Mimms EN6 3LD, UK;
DNA Damage Response Laboratory, London Research Institute, Cancer Research UK, Clare Hall, South Mimms EN6 3LD, UK;
Department of Radiation Oncology, Dana Farber Cancer Institute, Boston, Massachusetts 02215, USA;
DNA Damage Response Laboratory, London Research Institute, Cancer Research UK, Clare Hall, South Mimms EN6 3LD, UK;
Protein Analysis and Proteomics Laboratory, London Research Institute, Cancer Research UK, Clare Hall, South Mimms EN6 3LD, UK,Mass Spectrometry, Medical Research Council Laboratory of Molecular Biology, Hills Road, Cambridge CB2 0QH, UK;
Experimental Histopathology Laboratory, London Research Institute, Cancer Research UK, 44 Lincoln's Inn Fields, London WC2A 3LY, UK;
Department of Radiation Oncology, Dana Farber Cancer Institute, Boston, Massachusetts 02215, USA;
Institute of Molecular Cancer Research, University of Zurich, Winterthurerstrasse 190, CH-8057 Zurich, Switzerland;
Translational Cancer Therapeutics Laboratory, London Research Institute, Cancer Research UK, 44 Lincoln's Inn Fields, London WC2A 3LY, UK,UCL Cancer Institute, Huntley Street, London WC1E 6DD, UK;
DNA Damage Response Laboratory, London Research Institute, Cancer Research UK, Clare Hall, South Mimms EN6 3LD, UK;
机译:在p53缺陷的CHO细胞中,哺乳动物Rad51的过表达不会刺激肿瘤发生,而显性阴性的Rad51影响中心体片段化,倍性并刺激肿瘤发生。
机译:RAD51的高表达促进KRAS突变肺癌细胞中DNA损伤的修复和存活
机译:H3K36三甲基化介导的表观遗传调控被果蝇激活,并在果蝇早期卵子发育过程中促进生殖细胞分化H3K36三甲基化介导的表观遗传调控被果蝇激活,并在果蝇早期卵子发生期间促进生殖细胞分化。H3K36三甲基化介导的表观遗传调控被果蝇激活。并促进果蝇早期卵子发生过程中的生殖细胞分化
机译:人胚胎干细胞衍生的间充质干细胞和BMP7促进软骨修复
机译:DNA不匹配修复机制,调控及其在肿瘤发生中的作用的细胞方面
机译:HELQ促进RAD51旁系物依赖性修复避免生殖细胞磨损和肿瘤发生
机译:HELQ促进RAD51旁系同源蛋白修复,从而避免生殖细胞丢失和肿瘤发生
机译:剂量率对小鼠不同生殖细胞阶段辐射诱导突变频率的不同影响及其对肿瘤发生分析的启示