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机译:被动和主动塑造海马CA3锥体细胞的联合/合纵和穿孔路径突触的整体反应
Department of Neuroscience, University of Pittsburgh, Pittsburgh, PA, USA,Department of Otolaryngology, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA;
Center for Neural Informatics, Structure, & Plasticity, and Molecular Neuroscience Department, Krasnow Institute for Advanced Study, George Mason University, 4400 University Drive, MS 2A1, Fairfax, VA 22030, USA;
Center for Neural Informatics, Structure, & Plasticity, and Molecular Neuroscience Department, Krasnow Institute for Advanced Study, George Mason University, 4400 University Drive, MS 2A1, Fairfax, VA 22030, USA;
Department of Statistics, University of Pittsburgh, Pittsburgh, PA, USA;
Center for Neural Informatics, Structure, & Plasticity, and Molecular Neuroscience Department, Krasnow Institute for Advanced Study, George Mason University, 4400 University Drive, MS 2A1, Fairfax, VA 22030, USA;
Department of Neuroscience, University of Pittsburgh, Pittsburgh, PA, USA;
AMPA receptor; NMDA receptor; hippocampus;
机译:计算机模型的海马CA3锥体细胞的关联/连合和穿孔路径突触的统一反应。
机译:在新型TGF344-Alzheimer疾病大鼠大鼠模型中,在Schaffer Collateral-Ca1金字塔蛋白突触之前发生突触功能的缺陷发生在内侧穿孔路径牙齿颗粒细胞突触
机译:在新型TGF344-Alzheimer疾病大鼠模型中Schaffer壳体-CA1金字塔模型之前,“突触函数缺陷发生的突触函数缺陷发生的静脉曲张术”[2018年2月的疾病1月110日,第166-179页 ]
机译:从主动和被动成分方面分析单个心脏细胞的场致跨膜电位反应
机译:催产素持续改变Ca2金字塔细胞活性,从而形成海马神经递质
机译:来自关联/连持子和穿孔路径突触在海马CA3金字塔细胞中的无线和主动整形
机译:来自关联/连持子和穿孔路径突触在海马CA3金字塔细胞中的无线和主动整形