Corrigendum to “Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model” [Neurobiology of Disease Volume 110, February 2018, Pages 166–179]

Lindsey A. Smith; Lori L. McMahon

首页> 外文期刊>Neurobiology of disease >Corrigendum to “Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model” [Neurobiology of Disease Volume 110, February 2018, Pages 166–179]

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Corrigendum to “Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model” [Neurobiology of Disease Volume 110, February 2018, Pages 166–179]

机译：在新型TGF344-Alzheimer疾病大鼠模型中Schaffer壳体-CA1金字塔模型之前，“突触函数缺陷发生的突触函数缺陷发生的静脉曲张术”[2018年2月的疾病1月110日，第166-179页 ]

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来源
《Neurobiology of disease》 |2018年第2018期|共2页
作者
Lindsey A. Smith; Lori L. McMahon;
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作者单位

Department of Cell Developmental and Integrative Biology University of Alabama at Birmingham;

Department of Cell Developmental and Integrative Biology University of Alabama at Birmingham;

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原文格式 PDF
正文语种 eng
中图分类神经病学与精神病学;
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1. Corrigendum to “Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model” [Neurobiology of Disease Volume 110, February 2018, Pages 166–179] [J] . Lindsey A. Smith, Lori L. McMahon Neurobiology of disease . 2018,第期

机译：在新型TGF344-Alzheimer疾病大鼠模型中Schaffer壳体-CA1金字塔模型之前，“突触函数缺陷发生的突触函数缺陷发生的静脉曲张术”[2018年2月的疾病1月110日，第166-179页 ]
2. Synaptic fatigue at the naive perforant path-dentate granule cell synapse in the rat. [J] . Abrahamsson T, Gustafsson B, Hanse E The Journal of Physiology . 2005,第Pta3期

机译：大鼠幼稚穿孔路径齿状颗粒细胞突触的突触疲劳。
3. Increased long-term potentiation at medial-perforant path-dentate granule cell synapses induced by selective inhibition of histone deacetylase 3 requires Fragile X mental retardation protein [J] . Aimee V. Franklin, James R. Rusche, Lori L McMahon Neurobiology of learning and memory . 2014,第Null期

机译：通过选择性抑制组蛋白脱乙酰基酶3诱导的内侧穿孔路径齿状颗粒突触的长期增强作用需要脆弱的X型智力障碍蛋白
4. Deficits in synaptic function occur at medial perforant path-granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer’s Disease Rat Model [O] . Lindsey A. Smith, Lori L. McMahon -1

机译：在新型TgF344-阿尔茨海默氏病大鼠模型中先于Schaffer侧支CA1锥体细胞突触的先于内侧穿孔路径-颗粒细胞突触发生突触功能的缺陷
5. Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model [O] . Lindsey A. Smith, Lori L. McMahon 2018

机译：在新型TGF344-Alzheimer疾病大鼠疾病大鼠模型中，突触函数突触函数中的突触函数缺陷发生在中介穿孔路径牙齿颗粒细胞突触

Corrigendum to “Deficits in synaptic function occur at medial perforant path-dentate granule cell synapses prior to Schaffer collateral-CA1 pyramidal cell synapses in the novel TgF344-Alzheimer's Disease Rat Model” [Neurobiology of Disease Volume 110, February 2018, Pages 166–179]

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