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机译:减少人类T型淋巴病毒I型(HTLV-I)税收特异性细胞毒性T淋巴细胞中Tim-3表达的HTLV-I感染。
Division of Molecular Pathology, Center for Chronic Viral Diseases;
Division of Molecular Pathology, Center for Chronic Viral Diseases;
Department of Neurology and Geriatrics, Graduate School of Medical and Dental Sciences, Kagoshima University, 8-35-1 Sakuragaoka, Kagoshima, Japan;
Department of Neurology and Geriatrics, Graduate School of Medical and Dental Sciences, Kagoshima University, 8-35-1 Sakuragaoka, Kagoshima, Japan;
Department of Immunology, Graduate School and Faculty of Medicine, University of the Ryukyus, Uehara 207, Nishihara-cho, Okinawa, Japan;
Department of Neurology and Geriatrics, Graduate School of Medical and Dental Sciences, Kagoshima University, 8-35-1 Sakuragaoka, Kagoshima, Japan;
Division of Molecular Pathology, Center for Chronic Viral Diseases;
机译:在人I型T淋巴细胞病毒(HTLV-1)中,在HTLV-1感染中税收特异性的细胞毒性T淋巴细胞上的Tim-3表达降低。
机译:HTLV-1感染中HTLV-1税收特异性细胞毒性T淋巴细胞上Tim-3的表达降低
机译:西非农村人口中的人类I型T淋巴细胞病毒(HTLV-1)前病毒负荷的定量:人类2型免疫缺陷病毒的发病机理没有增强,但是HTLV-1 I病毒原负荷与死亡率有关。
机译:CD4 + T细胞HTLV-I感染的确定性和随机模型
机译:I型人T细胞淋巴病毒(HTLV-1)通过NF-κB和干扰素调节元件激活IL-15Ralpha表达。
机译:HTLV-1感染中HTLV-1税收特异性细胞毒性T淋巴细胞上Tim-3表达的降低
机译:HTLV-1感染中HTLV-1税收特异性细胞毒性T淋巴细胞上Tim-3表达的降低