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机译:Tip60的部分丢失在脊髓小脑共济失调1型(SCA1)小鼠模型中减缓了中期神经变性
Institute of Human Genetics and Institute of Translational Neuroscience,|Department of Biochemistry, Biophysics and Molecular Biology and;
Institute of Human Genetics and Institute of Translational Neuroscience,|Department of Laboratory Medicine and Pathology, University of Minnesota, Minneapolis, MN 55455, USA,;
Institute of Human Genetics and Institute of Translational Neuroscience,;
Department of Cell Biology, Neurobiology, and Anatomy, Medical College of Wisconsin, Milwaukee, WI 53226,;
Department of Molecular and|Department of Human Genetics, Pediatrics, and Howard Hughes Medical Institute, Baylor College of Medicine, Houston, TX 77030, USA;
机译:Tip60的部分丢失会减慢脊髓小脑共济失调1型(SCA1)小鼠模型的中期神经变性。
机译:氨基吡啶纠正1型脊髓小脑共济失调小鼠模型的早期功能障碍和延迟神经变性。
机译:在1型脊髓小脑共济失调小鼠模型中无症状地检测症状前和进行性神经退行性变。
机译:EMG矫正对2型脊髓小脑共济失调(SCA2)的皮质肌和肌间相干估计的作用。
机译:婴儿起病的脊髓小脑共济失调13型突变导致发展和快速退化的表型与浦肯野细胞过度兴奋有关。
机译:Tip60的部分丢失在脊髓小脑共济失调1型(SCA1)小鼠模型中减缓了中期神经变性
机译:Tip60的部分丢失在脊髓小脑共济失调1型(SCA1)小鼠模型中减缓了中期神经变性