机译:CSC-3436通过ROS介导的P38 / Chec /死亡受体5发信号通路对三重阴性乳腺癌细胞进行三重阴性乳腺癌细胞以逐行诱导的细胞凋亡
Department of Biological Science and Technology College of Life Sciences China Medical University Taichung Taiwan;
Department of Biological Science and Technology College of Life Sciences China Medical University Taichung Taiwan;
Division of Neurosurgery Buddhist Tzu Chi General Hospital Taichung Branch Taichung Taiwan School of Medicine Tzu Chi University Hualien Taiwan Department of Medical Imaging and Radiological Science Central Taiwan University of Science and Technology Taichung Taiwan;
Biotechnology Industry College of Life Sciences China Medical University Taichung Taiwan;
Department of Food Science Rutgers University New Brunswick New Jersey USA;
Department of Neurosurgery E-Da Hospital/Ⅰ-Shou University Kaohsiung Taiwan School of Medicine Ⅰ-Shou University Kaohsiung Taiwan;
Department of Biological Science and Technology College of Life Sciences China Medical University Taichung Taiwan Biotechnology Industry College of Life Sciences China Medical University Taichung Taiwan Department of Health and Nutrition Biotechnology Asia University Taichung Taiwan;
CSC-3436; death receptor; ROS; TARIL; triple negative breast cancer;
机译:Ilimaquinone诱导死亡受体表达,并通过激活ROS-ERK / p38 MAPK-CHOP信号通路使人结肠癌细胞对TRAIL诱导的细胞凋亡敏感
机译:活体内通过诱导死亡受体,在抗性三重阴性乳腺癌细胞中诱导死亡受体和细胞存活蛋白的下调,增强了诱导的细胞凋亡
机译:激活转录因子4(ATF4)-ATF3-C / EBP同源蛋白(CHOP)级联在四氯苯醌挑战的ER应激诱导的PC12细胞通过死亡受体5(DR5)信号介导的ROS介导的凋亡中显示出重要作用。
机译:TNF siRNA作为细胞凋亡和血管生成的调节剂,在三重阴性乳腺癌细胞中的细胞迁移和侵袭中(TNBC)
机译:使用DNA片段测定和细胞死亡染料验证NBA光动力学治疗后MDA-MB-231三阴性乳腺癌细胞的凋亡
机译:physagulide P:ROS / JNK信号通路对三阴性乳腺癌细胞的抗增殖诱导凋亡和自噬细胞死亡
机译:激活转录因子4(ATF4)-ATF3-C / EBP同源蛋白(Chec)级联显示出在ER应激诱导的四氯醌挑战的PC12细胞对ROS介导的凋亡中通过死亡受体5(DR5)信号传导的重要作用