机译:白藜芦醇通过抑制P38MapK,NF-κB信号传导和NLRP3炎性组活化来保护人支气管上皮细胞免受镍诱导的毒性。
School of Life Science Liaoning University Shenyang China;
School of Life Science Liaoning University Shenyang China;
School of Life Science Liaoning University Shenyang China;
Department of Mine Metallurgy and Geology Engineering University of Guanajuato Guanajuato Mexico;
School hospital Liaoning University Shenyang China;
School of Life Science Liaoning University Shenyang China;
School of Life Science Liaoning University Shenyang China;
inflammation; nickel; oxidative stress; resveratrol; ROS;
机译:白藜芦醇通过抑制P38MapK,NF-Kappa发信号和NLRP3炎症组活化来保护人支气管上皮细胞免受镍诱导的毒性。
机译:咖啡因通过抑制LPS诱导的THP-1巨噬细胞中的MAPK / NF-κB和A2AR信号传导来抑制NLRP3炎症组活化
机译:eon药苷通过p38 MAPK,ERK和NF-κB信号通路上调人支气管上皮细胞中β-防御素2的表达。
机译:表没食子儿茶素没食子酸酯抑制佛波酯诱导的NF-κB和CREB活化在小鼠皮肤中的作用p38 MAPK
机译:调节人白细胞介素17A,白细胞介素27,肿瘤坏死因子-α和人类嗜碱性粒细胞在炎性疾病中激活人支气管上皮细胞的细胞内信号转导机制
机译:白藜芦醇通过抑制NF-κB和p38 MAPK信号通路的激活逆转P-糖蛋白介导的U2OS / ADR细胞多药耐药性
机译:通过ROS介导的MAPK,NF-κB信号传导途径和NLRP3炎症烹饪人支气管上皮细胞对人支气管上皮细胞的毒性