机译:GADD45a对人喹喹诱导的人肝癌G2细胞凋亡的影响:线粒体功能障碍
Department of Pharmacology and Toxicology, College of Veterinary Medicine, China Agricultural University, Yuanmingyuan West Road No. 2, Haidian District, Beijing, PR China;
Department of Pharmacology and Toxicology, College of Veterinary Medicine, China Agricultural University, Yuanmingyuan West Road No. 2, Haidian District, Beijing, PR China;
Department of Pharmacology and Toxicology, College of Veterinary Medicine, China Agricultural University, Yuanmingyuan West Road No. 2, Haidian District, Beijing, PR China;
Department of Pharmacology and Toxicology, College of Veterinary Medicine, China Agricultural University, Yuanmingyuan West Road No. 2, Haidian District, Beijing, PR China;
Department of Pharmacology and Toxicology, College of Veterinary Medicine, China Agricultural University, Yuanmingyuan West Road No. 2, Haidian District, Beijing, PR China;
Department of Pharmacology and Toxicology, College of Veterinary Medicine, China Agricultural University, Yuanmingyuan West Road No. 2, Haidian District, Beijing, PR China;
Department of Pharmacology and Toxicology, College of Veterinary Medicine, China Agricultural University, Yuanmingyuan West Road No. 2, Haidian District, Beijing, PR China;
Olaquindox; GADD45a; Apoptosis; Mitochondrial dysfunction; ROS;
机译:GADD45a通过JNK / p38途径调节人肝癌G2细胞中喹乙醇诱导的DNA损伤和S期阻滞。
机译:Gadd45a通过JNK / P38途径调节人肝癌G2细胞中的Olaquindox诱导的DNA损伤和S相阻滞
机译:新的类黄酮在人肝癌HepG2细胞中诱导的凋亡涉及活性氧介导的线粒体功能障碍和MAPK活化。
机译:杨梅素诱导人肝癌HepG2细胞凋亡和G2 / M细胞周期阻滞
机译:二十碳五烯酸(EPA)通过p53途径诱导人肝癌细胞凋亡。
机译:GADD45a通过JNK / p38途径调节人肝癌G2细胞中由喹乙醇引起的DNA损伤和S期阻滞。
机译:GaDD45a通过JNK / p38途径调节喹乙醇诱导的人肝癌G2细胞DNa损伤和s期阻滞