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机译:肝来源的外来体lncRNA MT1 DP加剧了镉诱导的肾毒性
Chinese Acad Sci Res Ctr Ecoenvironm Sci State Key Lab Environm Chem & Ecotoxicol Beijing 100085 Peoples R China|Univ Chinese Acad Sci Beijing 100049 Peoples R China;
Southeast Univ Sch Publ Hlth Minist Educ Key Lab Environm Med Engn Nanjing 210009 Jiangsu Peoples R China;
Chinese Acad Sci Res Ctr Ecoenvironm Sci State Key Lab Environm Chem & Ecotoxicol Beijing 100085 Peoples R China|Capital Med Univ Beijing Friendship Hosp Liver Res Ctr Beijing 100050 Peoples R China;
Guangdong Prov Ctr Dis Control & Prevent Guangzhou 511430 Peoples R China;
Guangdong Prov Ctr Dis Control & Prevent Guangzhou 511430 Peoples R China|Southern Med Univ Sch Publ Hlth Food Safety & Hlth Res Ctr Guangzhou 100022 Peoples R China;
Chinese Acad Med Sci China Natl Ctr Food Safety Risk Assessment Food Safety Res Unit 2019RU014 NHC Key Lab Food Safety Risk Assessment Beijing 100022 Peoples R China;
Southern Med Univ Sch Publ Hlth Food Safety & Hlth Res Ctr Guangzhou 100022 Peoples R China|Chinese Acad Med Sci China Natl Ctr Food Safety Risk Assessment Food Safety Res Unit 2019RU014 NHC Key Lab Food Safety Risk Assessment Beijing 100022 Peoples R China;
Cadmium; LncRNA; MT1DP; Nephrotoxicity; Exosomes;
机译:LncRNA MT1DP通过抑制Nrf2的功能加重镉诱导的氧化应激,并取决于与miR-365的相互作用
机译:葡萄糖醛酸化作用在N-(3,5-二氯苯基)琥珀酰亚胺(NDPS)肾毒性中的作用:NDPS和NDPS代谢产物在Gunn,Wistar和Fischer 344大鼠中的肾毒性潜力。
机译:组合制剂Hepeel〜®预防镉诱导的肝源性细胞毒性
机译:LncRNA MT1DP通过抑制Nrf2的功能加重镉诱导的氧化应激并取决于与miR-365的相互作用
机译:芯片揭示镉诱导人支气管上皮细胞恶性转化的LncRNAs表达特征