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机译:小碱通过激活Akt信号通路改善四氧嘧啶诱导的糖尿病C57BL / 6小鼠的高血糖
Laboratory of Pharmacology & Toxicology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China;
Chengdu Military General Hospital, Chengdu 610083, China;
Laboratory of Pharmacology & Toxicology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China;
Laboratory of Pharmacology & Toxicology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China;
Laboratory of Pharmacology & Toxicology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China;
Laboratory of Pharmacology & Toxicology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China;
Laboratory of Pharmacology & Toxicology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China;
Laboratory of Pharmacology & Toxicology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China;
Laboratory of Pharmacology & Toxicology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China;
Laboratory of Pharmacology & Toxicology, School of Pharmaceutical Sciences, Sun Yat-Sen University, Guangzhou 510006, China;
diabetic mouse; berberine; akt signaling pathway; GSK-3P; Glucokinase;
机译:通过AKT信号通路激活AKT信号通路,豆芽蛋白在阿昔班诱导的糖尿病C57BL / 6小鼠中改善高血糖症
机译:小碱可改善糖尿病C57BL / 6小鼠的肾脏损伤:抑制SphK-S1P信号通路
机译:当归通过激活AMP激活的蛋白激酶信号通路来改善C57BL / KsJ-db / db小鼠的高血糖和肝脂肪变性
机译:糖尿病小鼠抗黄芪抗氧化氧胁迫的多糖对糖尿病小鼠的保护作用
机译:670nm近红外光处理的光生物调节改善了C57BL / 6小鼠MOG35-55诱导的实验性自身免疫性脑脊髓炎(EAE)的疾病严重程度。
机译:Timosaponin B-II通过四氧嘧啶诱导的小鼠中的TXNIPmTOR和NF-κB信号通路改善糖尿病性肾病
机译:通过AKT信号通路激活AKT信号通路,豆芽蛋白在阿昔班诱导的糖尿病C57BL / 6小鼠中改善高血糖症