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机译:下丘脑IGF-1 / PI3K / Akt / mTOR信号通路介导的DEHP对女性性早熟的干扰
Jilin Univ, China Japan Union Hosp, Sci Res Ctr, Changchun, Jilin, Peoples R China;
Jilin Univ, China Japan Union Hosp, Sci Res Ctr, Changchun, Jilin, Peoples R China|Jilin Univ, Sch & Hosp Stomatol, Dept Orthodont, Changchun, Jilin, Peoples R China;
Jilin Univ, China Japan Union Hosp, Sci Res Ctr, Changchun, Jilin, Peoples R China;
Jilin Univ, China Japan Union Hosp, Sci Res Ctr, Changchun, Jilin, Peoples R China;
Jilin Univ, China Japan Union Hosp, Sci Res Ctr, Changchun, Jilin, Peoples R China;
Jilin Univ, China Japan Union Hosp, Gastrointestinal & Colorectal Surg, Changchun, Jilin, Peoples R China;
Jilin Univ, Sch & Hosp Stomatol, Dept Orthodont, Changchun, Jilin, Peoples R China;
Jilin Univ, China Japan Union Hosp, Sci Res Ctr, Changchun, Jilin, Peoples R China;
Jilin Univ, China Japan Union Hosp, Sci Res Ctr, Changchun, Jilin, Peoples R China;
Di-(2-ethylhexyl) phthalate; Female precocious puberty; Hypothalamus; IGF-1/PI3K/Akt/mTOR signaling pathway;
机译:下丘脑IGF-1 / PI3K / MTOR信号通路介导的雌性雌性Dehp的干扰
机译:IGF-1通过激活胰腺癌细胞中的IGF-1 / PI3K / Akt信号通路来介导PTEN抑制并增强细胞侵袭和增殖。
机译:前颗粒蛋白可通过PI3K / Akt / mTOR途径补偿IGF-1信号通路受阻,从而促进C2C12成肌细胞的肌肥大
机译:无标记的磷蛋白质方法,用于研究新型PI3K / MTOR抑制剂在癌细胞中的信号通路
机译:PI3k / AKT / mTOR信号通路的遗传分析。
机译:类黄酮通过下调PI3Kγ介导的PI3K / AKT / mTOR / p70S6K / ULK信号通路抑制人乳腺癌细胞的增殖并诱导细胞凋亡和自噬
机译:前颗粒蛋白可通过PI3K / Akt / mTOR途径补偿IGF-1信号传导受阻,从而促进C2C12成肌细胞中的肌管肥大