机译:MiR-33a调节胰岛中的ABCA1表达,胆固醇蓄积和胰岛素分泌
Department of Medical Genetics, Centre for Molecular Medicine and Therapeutics, Child and Family Research Institute, University of British Columbia, Vancouver, British Columbia, Canada;
Department of Medical Genetics, Centre for Molecular Medicine and Therapeutics, Child and Family Research Institute, University of British Columbia, Vancouver, British Columbia, Canada;
Department of Medical Genetics, Centre for Molecular Medicine and Therapeutics, Child and Family Research Institute, University of British Columbia, Vancouver, British Columbia, Canada;
Institute for Heart and Lung Health, St. Paul's Hospital, Vancouver, British Columbia, Canada;
Department of Medical Genetics, Centre for Molecular Medicine and Therapeutics, Child and Family Research Institute, University of British Columbia, Vancouver, British Columbia, Canada;
Department of Surgery, Child and Family Research Institute, University of British Columbia, Vancouver, British Columbia, Canada;
Department of Surgery, Child and Family Research Institute, University of British Columbia, Vancouver, British Columbia, Canada,Depart-ment of Pathology and Laboratory Medicine, Child and Family Research Institute, University of British Columbia, Vancouver, British Columbia, Canada;
Department of Medical Genetics, Centre for Molecular Medicine and Therapeutics, Child and Family Research Institute, University of British Columbia, Vancouver, British Columbia, Canada;
机译:来自OCimum物种的酚类物质增强了葡萄糖刺激的胰岛素分泌并调节小鼠胰岛中关键胰岛素调控基因的表达
机译:由于ABCA1丢失导致胰岛胆固醇积聚导致胰岛素颗粒的胞吐功能受损。
机译:信标基因在大鼠胰岛中的表达:信标(47-73)蛋白(泛素样蛋白5)对胰岛素的体内分泌和孤立的胰岛释放胰岛素的作用相反。
机译:亚砷酸盐及其代谢物,甲基胂岩,抑制胰岛胰岛血糖胰岛素分泌期间的钙流量
机译:囊性纤维化跨膜电导调节剂在胰岛β细胞胰岛素分泌中的作用。
机译:miR-33a调节胰岛中的ABCA1表达胆固醇蓄积和胰岛素分泌
机译:由于aBCa1缺失导致胰岛胆固醇累积导致胰岛素颗粒的胞吐作用受损