机译:蛋白酪氨酸磷酸酶的失活增强胰岛中的干扰素信号传导。
St Vincent's Institute of Medical Research, Melbourne, Victoria, Australia,Department of Medicine, St Vincent's Hospital, The University of Melbourne, Melbourne, Victoria, Australia;
St Vincent's Institute of Medical Research, Melbourne, Victoria, Australia;
St Vincent's Institute of Medical Research, Melbourne, Victoria, Australia,Department of Medicine, St Vincent's Hospital, The University of Melbourne, Melbourne, Victoria, Australia;
Diabetic Complications Division, Baker IDI Heart and Diabetes Institute, Melbourne, Victoria, Australia;
St Vincent's Institute of Medical Research, Melbourne, Victoria, Australia,Department of Medicine, St Vincent's Hospital, The University of Melbourne, Melbourne, Victoria, Australia;
Department of Biochemistry and Molecular Biology, Monash University, Melbourne, Victoria, Australia;
Diabetic Complications Division, Baker IDI Heart and Diabetes Institute, Melbourne, Victoria, Australia;
St Vincent's Institute of Medical Research, Melbourne, Victoria, Australia,Department of Medicine, St Vincent's Hospital, The University of Melbourne, Melbourne, Victoria, Australia;
St Vincent's Institute of Medical Research, Melbourne, Victoria, Australia,Department of Medicine, St Vincent's Hospital, The University of Melbourne, Melbourne, Victoria, Australia;
机译:组成型光学发生1蛋白与蛋白质 - 酪氨酸磷酸酶1B的相互作用抑制蛋白质 - 酪氨酸磷酸酶1B活性并增强胰岛素信号传导
机译:亲营养素信号通过使受体型蛋白酪氨酸磷酸酶β/ζ的内在催化活性失活而增加β-catenin的酪氨酸磷酸化
机译:木犀草素通过激活Janus激酶/信号转导子和转录途径信号转导子(通过蛋白激酶A介导的酪氨酸磷酸酶SHP-2的抑制作用)来激活干扰素α/β的抗增殖作用。
机译:通过蛋白质酪氨酸磷酸酶-1b的小竞争性抑制来提高双(乙酰丙酮酰基)氧化钒(IV)的增强
机译:饮食和内源性物质使蛋白质酪氨酸磷酸酶失活
机译:本构光形态发生1蛋白与蛋白酪氨酸磷酸酶1B的相互作用抑制蛋白酪氨酸磷酸酶1B活性并增强胰岛素信号传导。
机译:蛋白质酪氨酸磷酸酶的失活增强了胰岛中的干扰素信号传导。