机译:删除胎盘生长因子可预防糖尿病性视网膜病,并与Akt激活和HIF1α-VEGF途径抑制相关
Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD;
Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD,Guangxi Tumor Hospital and Institute, Nanning, Guangxi, China;
Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD;
Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD;
Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD,Aier Eye Hospital, Changsha, Hunan, China;
Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD,China Japan Union Hospital, Changchun, Jilin, China;
Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD;
Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD;
Wilmer Eye Institute, Johns Hopkins University School of Medicine, Baltimore, MD;
机译:MicroRNA-21的抑制通过PTEN依赖于PI3K / AKT / VEGF信号通路在糖尿病视网膜病变中抑制视网膜血管内皮细胞生长和血管生成
机译:阻断雷帕霉素(mTOR)的哺乳动物靶标可减弱糖尿病性视网膜病变中的HIF-1?±通路参与的血管内皮生长因子(VEGF)。
机译:在糖尿病性视网膜病的啮齿动物模型中,小分子干扰核糖核酸抑制结缔组织生长因子可防止细胞外基质分子增加
机译:基于透明质酸的多价VEGF拮抗剂抑制糖尿病性视网膜病变中的致病性血管生成。
机译:BP1通过激活VEGF调节PI3K / Akt途径。
机译:删除胎盘生长因子可预防糖尿病性视网膜病并与Akt激活和HIF1α-VEGF途径抑制相关
机译:丢失胎盘生长因子可防止糖尿病视网膜病变,与AKT激活和HIF1α-VEGF途径抑制相关。糖尿病2015; 64:200-212