机译:糖尿病逆转骨髓动员病和血管紧张素-(1-7)增强的血管修复
Department of Pharmaceutical Sciences, North Dakota State University, Fargo, ND;
Department of Pharmaceutical Sciences, North Dakota State University, Fargo, ND;
Department of Pharmacology and Nutritional Sciences, University of Kentucky, Lexington, KY;
BetaStem Therapeutics, Inc., San Francisco, CA;
Department of Pharmacology and Nutritional Sciences, University of Kentucky, Lexington, KY;
Department of Pharmaceutical Sciences, North Dakota State University, Fargo, ND;
机译:血管紧张素-(1-7)增强了血管紧张素II诱导的小鼠骨髓源性树突状细胞中ERK1 / 2的磷酸化。
机译:培养的人骨髓来源的CD31(+)细胞通过增强的血管生成,粘附和抗炎作用可有效用于心脏和血管修复
机译:过度表达人血管内皮生长因子165的骨髓基质细胞的移植可增强辐射诱发的大鼠模型的组织修复
机译:血管化的骨髓移植导致比常规细胞骨髓移植更高水平的造血逆变
机译:血管紧张素-(1-7):糖尿病干细胞运动和血管修复的目标。
机译:糖尿病逆转骨髓动员病和血管紧张素-(1-7)增强的血管修复
机译:骨髓骨髓病变的逆转,血管紧张素 - (1-7)中的血管修复在糖尿病中