Sodium bicarbonate ingestion in the general population is common, given that it is an antacid employed for dyspepsia and gastroesophageal reflux, but it has been associated with spontaneous gastric rupture. The first case was reported in 1842 by Thompson et al. 1 The phenomenon has not been widely studied, and the following are among the most common causes of spontaneous gastric perforation: abundant food ingestion by patients with psychiatric dis- orders (anorexia nervosa and bulimia), sodium bicarbonate ingestion, superior mesenteric artery syndrome, gastric infarction, strangulated hiatal hernia, gastric volvulus, and trauma. Because it is a pathology with a high mortality rate, it requires immediate intervention. Thus, emphasizing the existence of these cases is of the utmost importance.
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