Experimental Study of Correction of Endothelial Dysfunction Solutions Ultra-Low Doses of Antibodies.

摘要

ABSTRACT Modeling deficit of nitric oxide L-NAME administration to rats resulted in the development of hypertension and endothelial dysfunction. Under this model expressed endothelium and cardioprotective properties of ultra-low doses of antibodies to the endothelial NO- synthase, VEGF and c- terminal fragment of the AT1 angiotensin II have been identified. The results of this study allow us to recommend the use of ultralow doses of antibodies to endothelial NO- synthase, VEGF and c- terminal f.