The effect of viable Chlamydia pneumoniae on serum cytokines and adhesion molecules in hemodialysis patients

摘要

The effect of viable Chlamydia pneumoniae on serum cytokines and adhesion molecules in hemodialysis patients.BackgroundChlamydia pneumoniae (Cp) induces the production of cytokines and adhesion molecules in infected host eukaryotic cells. The causes for pro-inflammatory cytokine and adhesion molecule increase in hemodialysis (HD) patients have not been fully elucidated. The possibility that, in this particularly atherosclerotic population, Cp, a microorganism implicated in the infectious-based inflammatory hypothesis of atherosclerosis' is also responsible for these molecules' increase is assessed in this study.