A KSHV microRNA Directly Targets G Protein-Coupled Receptor Kinase 2 to Promote the Migration and Invasion of Endothelial Cells by Inducing CXCR2 and Activating AKT Signaling

Brian J. Krueger; Chun Lu; Cong Wang; Di Qin; Jianzhong Zhu; Minmin Hu; Qin Yan; Rolf Renne; Shou-Jiang Gao; Wan Li; Weiping Lu; Zhiqiang Bai

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A KSHV microRNA Directly Targets G Protein-Coupled Receptor Kinase 2 to Promote the Migration and Invasion of Endothelial Cells by Inducing CXCR2 and Activating AKT Signaling

机译：KSHV microRNA直接靶向G蛋白偶联的受体激酶2，通过诱导CXCR2和激活AKT信号来促进内皮细胞的迁移和侵袭。

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《PLoS Pathogens》 |2015年第9期|共页
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Brian J. Krueger; Chun Lu; Cong Wang; Di Qin; Jianzhong Zhu; Minmin Hu; Qin Yan; Rolf Renne; Shou-Jiang Gao; Wan Li; Weiping Lu; Zhiqiang Bai;
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中图分类生物科学;
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1. Stromal Cell-Derived Factor-1 Promotes Endothelial Colony-Forming Cell Migration Through the Ca2+-Dependent Activation of the Extracellular Signal-Regulated Kinase 1/2 and Phosphoinositide 3-Kinase/AKT Pathways [J] . Zuccolo Estella, Di Buduo Christian, Lodola Francesco, Stem cells and development . 2018,第1期

机译：基质细胞衍生的因子-1通过细胞外信号调节激酶1/2和磷酸亚膦酰化3-激酶/ Akt途径的Ca2 +依赖性活化促进内皮菌落形成细胞迁移
2. Activation of liver X receptor enhances the proliferation and migration of endothelial progenitor cells and promotes vascular repair through PI3K/Akt/eNOS signaling pathway activation [J] . Yu Jie, Wang Qiang, Wang Hang, Vascular pharmacology . 2014,第3期

机译：肝脏X受体的激活通过PI3K / Akt / eNOS信号通路激活增强内皮祖细胞的增殖和迁移并促进血管修复
3. CXCL5/CXCR2 axis promotes bladder cancer cell migration and invasion by activating PI3K/AKT-induced upregulation of MMP2/MMP9 [J] . Ye Gao, Zhenfeng Guan, Jiaqi Chen, International journal of oncology . 2015,第2期

机译：CXCL5 / CXCR2轴通过激活PI3K / AKT诱导的MMP2 / MMP9上调来促进膀胱癌细胞的迁移和侵袭
4. Role of G proteins and the p38 Mitogen Activated Protein (MAP) kinase in Lipopolysaccharide (LPS)- induced signaling in human endothelial cells (EC) [C] . Moshe Arditi. Aninda Das, Yiping Jin NATO Advanced Study Institute on Vascular Endothelium : Mechanisms of Cell Signaling . 1999

机译：G蛋白和P38丝裂原活化蛋白（MAP）激酶在人内皮细胞（EC）中的信号传导中的G蛋白和P38丝裂原激活蛋白（MAP）激酶的作用
5. The multi-targeted receptor tyrosine kinase inhibitor, linifanib (ABT-869), induces apoptosis and inhibits proliferation of leukemia cells through phosphoinositide-3 kinase (PI3K)/AKT-dependent signaling pathways. [D] . Hernandez, Jenny Elizabeth. 2010

机译：多靶点受体酪氨酸激酶抑制剂利尼法尼（ABT-869）通过磷酸肌醇3激酶（PI3K）/ AKT依赖性信号传导途径诱导凋亡并抑制白血病细胞的增殖。
6. A KSHV microRNA Directly Targets G Protein-Coupled Receptor Kinase 2 to Promote the Migration and Invasion of Endothelial Cells by Inducing CXCR2 and Activating AKT Signaling [O] . Minmin Hu, Cong Wang, Wan Li, 2015

机译：KSHV microRNA直接靶向G蛋白偶联的受体激酶2通过诱导CXCR2和激活AKT信号来促进内皮细胞的迁移和侵袭。
7. A KSHV microRNA Directly Targets G Protein-Coupled Receptor Kinase 2 to Promote the Migration and Invasion of Endothelial Cells by Inducing CXCR2 and Activating AKT Signaling [O] . Hu M, Wang C, Li W, 2015

机译：KSHV microRNA直接靶向G蛋白偶联的受体激酶2，通过诱导CXCR2和激活AKT信号来促进内皮细胞的迁移和侵袭。

A KSHV microRNA Directly Targets G Protein-Coupled Receptor Kinase 2 to Promote the Migration and Invasion of Endothelial Cells by Inducing CXCR2 and Activating AKT Signaling

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