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Intestinal perforation after surgical treatment for incisional hernia: iatrogenic or idiopathic?

机译:切口疝手术治疗后的肠穿孔:医源性还是特发性?

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Intestinal perforation (IP) is a life-threatening gastroenterological condition requiring urgent surgical care, which may present itself as an uncommon complication following incisional hernia repair surgery, most often because of iatrogenic traumatism occurring during the procedure. However, we report a case where a spontaneous onset can be hypothesised. A 60-years-old patient underwent repair of an abdominal laparocele, through rectus abdominis muscle plasty, 5?years after development of an incisional hernia due to exploratory laparotomy for the treatment of acute appendicitis. Xipho-pubic scar was excised and umbilicus and supra-umbilical hernia sac dissected, a linear median incision was performed along the sub-umbilical linea alba, reaching preperitoneal plane to assess any intestinal loop adherence to the abdominal wall. After limited viscerolysis, abdominal wall defect was corrected by ‘rectus abdominis muscle plasty’ and umbilicus reconstruction by Santanelli technique. Postoperative course was uneventful until Day 29, with sudden onset of epigastric pain, fever and bulge. Sixty cubic centimeter pus was drained percutaneously and cavity was rinsed with a 50% H2O2 and H2O V-V solution until draining clear fluid. Symptoms recurred two days later, while during rinsing presented dyspnoea. X-Ray and CT scan diagnosed IP, and she underwent under emergency an exploratory laparotomy, leading to right hemicolectomy extended to last ileal loops and middle third of the transverse, right monolateral salpingo-ovariectomy and a temporary ileostomy by general surgeon. Twenty-three days later an ileostomy reversal surgery was performed and 8 days after she was discharged. At latest follow-up patient showed fair conditions, complaining abdominal pain and diarrhoea, attributable to the extensive intestinal resection. IP following incisional hernia repair, is reported as uncommon and early postoperative complication. In our case, the previous regular postoperative course with late onset lead us to hypothesise a possible idiopathic etiopathogenesis, because of a strangulation followed by gangrene and abscess formation, which might begin before the incisional hernia repair and unnoticed at the time surgery was performed.
机译:肠穿孔(IP)是危及生命的胃肠病,需要紧急外科护理,在切开疝修复手术后可能表现为罕见的并发症,通常是由于手术过程中发生的医源性创伤。但是,我们报告了一种可能自发发作的情况。一名60岁的患者因探查性剖腹术而导致切开性疝发展为急性阑尾炎后,通过腹直肌成形术进行了5年腹腹直肌修复术。切除耻骨耻骨下睑裂并切除脐带和脐上疝囊,沿脐下白线进行线性正中切口,到达腹膜前平面,以评估肠any对腹壁的粘附。有限的溶栓作用后,通过“腹直肌成形术”矫正腹壁缺损,并通过桑坦内利技术矫正脐带。直到第29天,术后病情平稳,并突然出现上腹痛,发烧和胀痛。经皮引流60立方厘米的脓液,并用50%的H2O2和H2O V-V溶液冲洗腔室,直到排出澄清的液体。两天后症状再次出现,而在冲洗过程中出现呼吸困难。 X射线和CT扫描诊断为IP,她在紧急情况下进行了探查性剖腹手术,导致右半结肠切除术延伸到最后的回肠loop和横向,右单侧输卵管卵巢切除术的中间三分之一,并由普通外科医师进行了临时性回肠造口术。 23天后进行回肠造口术逆转手术,出院后8天。在最新的随访中,患者表现出良好的状况,主诉腹部疼痛和腹泻,这归因于广泛的肠切除术。切开疝修补术后的IP报道为罕见和术后早期并发症。在我们的病例中,以前的常规术后晚期发作使我们假设可能存在特发性病因,原因是绞窄继发坏疽和脓肿形成,这可能在切开疝修复之前就开始了,并且在手术时未被发现。

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