In vivo rejection of tumor cells dependent on CD8 cells that kill independently of perforin and FasL

Sung-Hyung Lee; Erez Bar-Haim; Arthur Machlenkin; Ofir Goldberger; Ilan Volovitz; Ezra Vadai; Esther Tzehoval; Lea Eisenbach

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In vivo rejection of tumor cells dependent on CD8 cells that kill independently of perforin and FasL

机译：依赖于CD8细胞的肿瘤细胞的体内排斥反应独立于穿孔素和FasL杀死

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《Cancer gene therapy》 |2004年第4期|共1页
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Sung-Hyung Lee; Erez Bar-Haim; Arthur Machlenkin; Ofir Goldberger; Ilan Volovitz; Ezra Vadai; Esther Tzehoval; Lea Eisenbach;
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中图分类肿瘤学;
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1. In vivo rejection of tumor cells dependent on CD8 cells that kill independently of perforin and FasL [J] . Sung-Hyung Lee, Erez Bar-Haim, Arthur Machlenkin, Cancer gene therapy . 2004,第3期

机译：依赖于CD8细胞的肿瘤细胞的体内排斥反应独立于穿孔素和FasL杀死
2. Islet allograft rejection by contact-dependent CD8+ T cells: perforin and FasL play alternate but obligatory roles. [J] . Sleater M, Diamond AS, Gill RG American journal of transplantation: official journal of the American Society of Transplantation and the American Society of Transplant Surgeons . 2007,第8期

机译：胰岛同种异体移植物被接触依赖性CD8 + T细胞排斥：穿孔素和FasL扮演着交替但必须的角色。
3. Alloprimed CD8+ T cells regulate alloantibody and eliminate alloprimed B cells through perforin- and FasL-dependent mechanisms [J] . ZimmererJ.M., PhamT.A., WrightC.L., American journal of transplantation: official journal of the American Society of Transplantation and the American Society of Transplant Surgeons . 2014,第2期

机译：异源引发的CD8 + T细胞通过穿孔素和FasL依赖性机制调节异源抗体并消除异源引发的B细胞
4. Anti-gastric cancer active immunity induced by FasL/B7-1 genes modified tumor cells [C] . Shi-Ying Zheng, Dong Jiang, Jun Zhao, BioMedical Information Engineering, 2009. FBIE 2009 . 2009

机译：FasL / B7-1基因修饰的肿瘤细胞诱导的抗胃癌主动免疫
5. Loss of B7-H1 enhances the tumor rejection capabilities of murine anti-tumor CD8+ T cells. [D] . Saber, Rana. 2010

机译：B7-H1的丢失增强了鼠抗肿瘤CD8 + T细胞的肿瘤排斥能力。
6. Alloprimed CD8+ T cells Regulate Alloantibody and Eliminate Alloprimed B cells through Perforin- and FasL-dependent mechanisms [O] . J.M. Zimmerer, T.A. Pham, C.L. Wright, -1

机译：异源引发的CD8 + T细胞通过Perforin和FasL依赖性机制调节异源抗体并消除异源引发的B细胞
7. Islet Allograft Rejection by Contact-Dependent CD8+T cells: Perforin and FasL Play Alternate but Obligatory Roles [O] . M. Sleater, A. S. Diamond, R. G. Gill 2007

机译：胰岛同种异体移植物抑制通过接触依赖性CD8 + T细胞：穿孔素和FASL扮演交替但是强制性的角色

In vivo rejection of tumor cells dependent on CD8 cells that kill independently of perforin and FasL

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