机译:淋巴细胞迁移至发炎的泪腺是由血管细胞粘附分子-1 /α4{beta} 1整合素,外周淋巴结蛋白/ L-选择素和淋巴细胞功能相关的抗原-1粘附途径介导的。
From the Department of Pathology,Stanford University School of Medicine, Stanford, California|the Pathology and Laboratory Medicine Service,Department of Veterans Affairs, Palo Alto Health Care System, Palo Alto, California;
and the Department of Environmental Medicine,Kagoshima University Faculty of Medicine, Kagoshima, Japan;
From the Department of Pathology,Stanford University School of Medicine, Stanford, California|the Pathology and Laboratory Medicine Service,Department of Veterans Affairs, Palo Alto Health Care System, Palo Alto, California;
From the Department of Pathology,Stanford University School of Medicine, Stanford, California|the Pathology and Laboratory Medicine Service,Department of Veterans Affairs, Palo Alto Health Care System, Palo Alto, California;
机译:次级淋巴组织趋化因子(SLC)在流动下刺激整合素α4β7介导的淋巴细胞粘附至粘膜膜黏着蛋白细胞粘附分子1(MAdCAM-1)。
机译:趋化因子基质细胞衍生因子1α调节α4β7整联蛋白介导的淋巴细胞对粘膜膜黏着蛋白细胞粘附分子1和纤连蛋白的粘附。
机译:I域介导beta_2整合素淋巴细胞功能相关抗原1与Junctionalpha_L粘附分子A的Functionalpha_L相互作用。
机译:/ spl alpha // sub v // spl beta // sub 3 /整合素与RGD接枝聚乙烯的分子设计提供了内皮细胞粘附的估计
机译:靶向淋巴细胞功能相关抗原1(LFA-1)和细胞间粘附分子1(ICAM-1)相互作用,以防止HIV-1的性传播
机译:淋巴细胞迁移至发炎的泪腺是由血管细胞粘附分子-1 /α4β1整合素周围节点Addressin / L-选择素和与淋巴细胞功能相关的抗原-1粘附途径介导的。
机译:淋巴细胞迁移到发炎的泪腺均由血管细胞粘附分子-1 /α4β1整合蛋白,外周节点地址/ L-SELECTIN,以及淋巴细胞功能相关抗原-1粘附途径
机译:白细胞功能相关抗原-1配体细胞间粘附分子-1的T细胞受体/ CD3介导的静息人CD4 + TCell活化共刺激涉及延长的肌醇磷脂水解和持续增加的In