机译:内皮型一氧化氮合酶基因缺陷的小鼠表现出出生后骨形成,骨量减少以及成骨细胞成熟和活动缺陷的显着迟缓。
From the Department of Histochemistry,Imperial College School of Medicine, Hammersmith Campus, London, United Kingdom;
From the Department of Histochemistry,Imperial College School of Medicine, Hammersmith Campus, London, United Kingdom;
From the Department of Histochemistry,Imperial College School of Medicine, Hammersmith Campus, London, United Kingdom;
From the Department of Histochemistry,Imperial College School of Medicine, Hammersmith Campus, London, United Kingdom;
From the Department of Histochemistry,Imperial College School of Medicine, Hammersmith Campus, London, United Kingdom;
From the Department of Histochemistry,Imperial College School of Medicine, Hammersmith Campus, London, United Kingdom;
the Cardiovascular Research Center,Harvard Medical School, Charlestown, Massachusetts;
and the William Harvey Research Institute,St. Bartholomew’s and Royal London School of Medicine and Dentistry, London, United Kingdom;
From the Department of Histochemistry,Imperial College School of Medicine, Hammersmith Campus, London, United Kingdom;
机译:小鼠内皮一氧化氮合酶缺乏导致软骨细胞增殖减少和软骨内骨生长
机译:小鼠内皮一氧化氮合酶缺乏导致软骨细胞增殖减少和软骨内骨生长
机译:内皮型一氧化氮合酶基因敲除小鼠的小动脉密度降低是由于高血压引起的,而不是内皮型一氧化氮合酶的组成型缺陷。
机译:调节内皮一氧化氮合酶(ENOS)活性和偶联在体外冲击波型裂解中的影响(ESWL)
机译:在子宫内缺乏血管内一氧化氮合酶的转基因小鼠中血管功能的编程。
机译:内皮型一氧化氮合酶基因缺陷的小鼠表现出出生后骨形成骨量减少以及成骨细胞成熟和活动缺陷的显着迟缓。
机译:内皮型一氧化氮合酶基因缺陷的小鼠表现出出生后骨形成,骨量减少以及成骨细胞成熟和活动缺陷的显着迟缓。