机译:巨噬细胞集落刺激因子动员的骨髓源性CXCR4 +细胞参与减少小鼠梗死面积并改善心肌梗死后的心脏重塑
From the Department of Cardiovascular Medicine and Regeneration* and the Division of Cardiovascular Sciences, the Department of Organ Regeneration, Shinshu University Graduate School of Medicine, Matsumoto;
the Department of Cardiovascular Medicine, Shinshu University School of Health Sciences, Matsumoto;
the Cancer Chemotherapy Center, Japanese Foundation for Cancer Research, Tokyo;
and the Third Department of Internal Medicine,¶ National Defense Medical College, Saitama, Japan;
myocardial infarction (MI),monocyte/macrophage lineage;
机译:在大心肌梗死的猪模型中,从骨髓或粒细胞集落刺激因子激活的单采血液穿刺产品中冠状动脉内灌注自体单核细胞可能无法改善重塑,收缩功能,灌注或梗死面积。
机译:在大心肌梗死的猪模型中,从骨髓或粒细胞集落刺激因子激活的单采血液穿刺产品中冠状动脉内灌注自体单核细胞可能无法改善重塑,收缩功能,灌注或梗死面积
机译:骨髓源性单核细胞的冠状动脉内灌注消除了急性心肌梗死后不利的左心室重构:在急性心肌梗死(REPAIR-AMI)试验中,从富集祖细胞的重新灌注和梗死重塑中获得的见解。
机译:动员骨髓衍生的前体细胞在心肌梗死后减少心律失常 - 小鼠的研究
机译:皮质骨干细胞衍生的外泌体对心肌梗死再灌注损伤和心脏重塑的治疗作用的机制
机译:巨噬细胞集落刺激因子动员的骨髓源性CXCR4 +细胞参与减少小鼠梗死面积并改善心肌梗死后的心脏重塑
机译:巨噬细胞集落刺激因子动员的骨髓源性CXCR4 +细胞参与减少小鼠梗死面积并改善心肌梗死后的心脏重塑