机译:Skp2缺陷型小鼠的阻塞性肾病的肾脏损害减少。
From the Department of Biochemistry 1* and the First Department of Medicine, Hamamatsu University School of Medicine, Hamamatsu;
the Division of Developmental Genetics, Center for Translational and Advanced Animal Research on Human Diseases, Tohoku University Graduate School of Medicine, Miyagi;
and the Department of Molecular and Cellular Biology, Medical Institute of Bioregulation, Kyushu University, Fukuoka, Japan;
Obstructive Nephropathy,Skp2-Deficient Mice;
机译:在Skp2缺陷型小鼠中,阻塞性肾病的肾脏损害减少。
机译:S100A8 / A9促进阻塞性肾病的实质损伤和肾纤维化
机译:环氧氧酶2型的破坏加剧了阻塞性肾病的凋亡和肾损伤
机译:肾性糖皮质激素受体功能的年龄依赖性降低通过小鼠的膳食限制逆转
机译:金属硫蛋白对小鼠阿霉素肾病的保护作用及糖尿病肾病发展过程中的基因表达谱
机译:Skp2缺陷型小鼠的阻塞性肾病的肾脏损害减少。
机译:Skp2缺陷型小鼠的阻塞性肾病的肾脏损害减少。