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机译:诱导型一氧化氮合酶缺乏症损害基质金属蛋白酶9活性并破坏肝缺血/再灌注损伤中的白细胞迁移
From the Dumont-University of California at Los Angeles Transplant Center, Division of Liver and Pancreas Transplantation, Department of Surgery, David Geffen School of Medicine at University of California at Los Angeles, California;
From the Dumont-University of California at Los Angeles Transplant Center, Division of Liver and Pancreas Transplantation, Department of Surgery, David Geffen School of Medicine at University of California at Los Angeles, California;
From the Dumont-University of California at Los Angeles Transplant Center, Division of Liver and Pancreas Transplantation, Department of Surgery, David Geffen School of Medicine at University of California at Los Angeles, California;
From the Dumont-University of California at Los Angeles Transplant Center, Division of Liver and Pancreas Transplantation, Department of Surgery, David Geffen School of Medicine at University of California at Los Angeles, California;
From the Dumont-University of California at Los Angeles Transplant Center, Division of Liver and Pancreas Transplantation, Department of Surgery, David Geffen School of Medicine at University of California at Los Angeles, California;
机译:诱导型一氧化氮合酶缺乏会损害肝缺血/再灌注损伤中的基质金属蛋白酶9活性并破坏白细胞迁移。
机译:脂多糖刺激的实验性肝缺血再灌注损伤中肝组织诱导型一氧化氮合酶(iNOS)的表达和脂质过氧化作用:氨基胍的作用。
机译:瑞芬太尼预处理可通过诱导型一氧化氮合酶表达减少大鼠肝缺血-再灌注损伤。
机译:一氧化氮供体调节肝缺血再灌注损伤大鼠LTC4合成酶的mRNA表达
机译:一氧化氮和后处理:急性缺血再灌注损伤的心脏保护方法。
机译:诱导型一氧化氮合酶缺乏症损害基质金属蛋白酶9活性并破坏肝缺血/再灌注损伤中的白细胞迁移
机译:肝缺血/再灌注损伤中诱导型一氧化氮合酶缺乏症损害基质金属蛋白酶9活性并破坏白细胞迁移。