首页> 美国卫生研究院文献>The Journal of Biological Chemistry >Tumor necrosis factor receptor-associated factor-6 and ribosomal S6 kinase intracellular pathways link the angiotensin II AT1 receptor to the phosphorylation and activation of the IκB kinase complex in vascular smooth muscle cells.

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Tumor necrosis factor receptor-associated factor-6 and ribosomal S6 kinase intracellular pathways link the angiotensin II AT1 receptor to the phosphorylation and activation of the IκB kinase complex in vascular smooth muscle cells.

机译：肿瘤坏死因子受体相关因子6和核糖体S6激酶的细胞内途径将血管紧张素II AT1受体与血管平滑肌细胞中IκB激酶复合物的磷酸化和激活联系起来。

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期刊名称 The Journal of Biological Chemistry
作者
Priscilla Doyon; Marc J. Servant;
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年(卷),期 2011(286),12
年度 2011
页码 10888
总页数 1
原文格式 PDF
正文语种
中图分类分子生物学;
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专利

1. Tumor Necrosis Factor Receptor-associated Factor-6 and Ribosomal S6 Kinase Intracellular Pathways Link the Angiotensin II AT1 Receptor to the Phosphorylation and Activation of the IκB Kinase Complex in Vascular Smooth Muscle Cells [J] . Priscilla Doyon, Marc Servant The Journal of biological chemistry . 2010,第40期

机译：肿瘤坏死因子受体相关因子-6和核糖体S6激酶细胞内途径将血管紧张素II AT1受体连接到血管平滑肌细胞中IκB激酶复合物的磷酸化和活化
2. Inhibition of cyclic AMP-dependent kinase by expression of a protein kinase inhibitor/enhanced green fluorescent fusion protein attenuates angiotensin II-induced type 1 AT1 receptor mRNA down-regulation in vascular smooth muscle cells. [J] . Wang X, Murphy TJ Molecular pharmacology. . 1998,第3期

机译：通过蛋白激酶抑制剂/增强型绿色荧光融合蛋白的表达抑制环状AMP依赖性激酶可减弱血管紧张素II诱导的1型AT1受体mRNA在血管平滑肌细胞中的下调。
3. Novel Phosphorylation-dependent Ubiquitination of Tristetraprolin by Mitogen-activated Protein Kinase/Extracellular Signal-regulated Kinase Kinase Kinase 1 (MEKK1) and Tumor Necrosis Factor Receptor-associated Factor 2 (TRAF2) [J] . Yvonne Schichl, Ulrike Resch, Christof Lemberger, The Journal of biological chemistry . 2011,第44期

机译：通过促丝裂解蛋白激酶/细胞外信号调节激酶激酶激酶激酶激酶激酶激酶激酶激酶激酶激酶激酶激酶激酶激酶激酶激酶1（MEKK1）和肿瘤坏死因子受体相关因子2（TRAF2）的新型磷酸化依赖性
4. Activation of Mitogen-activated Protein Kinases and Protein Kinase B/akt Signaling By Oxidative Stress in Vascular Smooth Muscle Cells: involvement in Vascular Pathophysiology [C] . Ashok K. Srivastava, Nihar R. Pandey, Antoine Blanc International Society for Heart Research.Congress . 2004

机译：血管平滑肌细胞氧化应激激活丝裂剂活化蛋白激酶和蛋白激酶B / Akt信号传导：血管病理生理学中的参与
5. Mitogen-activated protein kinases mediate angiotensin II-induced upregulation of AT1 receptors in the brain of heart failure rats . [D] . Wei, Shunguang. 2007

机译：丝裂原激活的蛋白激酶介导血管紧张素II诱导的心力衰竭大鼠脑中AT1受体的上调。
6. Tumor Necrosis Factor Receptor-associated Factor-6 and Ribosomal S6 Kinase Intracellular Pathways Link the Angiotensin II AT1 Receptor to the Phosphorylation and Activation of the IκB Kinase Complex in Vascular Smooth Muscle Cells [O] . Priscilla Doyon, Marc J. Servant 2010

机译：肿瘤坏死因子受体相关因子6和核糖体S6激酶的细胞内通路将血管紧张素II AT1受体磷酸化并激活血管平滑肌细胞中IκB激酶复合物。
7. Tumor necrosis factor receptor-associated factor-6 and ribosomal S6 kinase intracellular pathways link the angiotensin II AT1 receptor to the phosphorylation and activation of the IκB kinase complex in vascular smooth muscle cells. [O] . Doyon, Priscilla, Servant, Marc J. 2011

机译：肿瘤坏死因子受体相关因子6和核糖体S6激酶的细胞内途径将血管紧张素II AT1受体与血管平滑肌细胞中IκB激酶复合物的磷酸化和激活联系起来。

Tumor necrosis factor receptor-associated factor-6 and ribosomal S6 kinase intracellular pathways link the angiotensin II AT1 receptor to the phosphorylation and activation of the IκB kinase complex in vascular smooth muscle cells.

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