Exercise mitigates sleep-loss-induced changes in glucose tolerance mitochondrial function sarcoplasmic protein synthesis and diurnal rhythms

摘要

Sleep loss has emerged as a risk factor for the development of impaired glucose tolerance. The mechanisms underpinning this observation are unknown; however, both mitochondrial dysfunction and circadian misalignment have been proposed. Because exercise improves glucose tolerance and mitochondrial function, and alters circadian rhythms, we investigated whether exercise may counteract the effects induced by inadequate sleep.