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Caffeine prevents hyperoxia-induced lung injury in neonatal mice through NLRP3 inflammasome and NF-κB pathway

机译：咖啡因通过NLRP3炎性体和NF-κB途径预防高氧血症对新生小鼠的肺损伤

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The concentration of caffeine in serum and expression of A R in lung. Caffeine treatment significantly increased the concentration of caffeine in serum and decreased the A R protein level in the lung. The concentration of caffeine in serum as determined by ELISA. The expression of A R in lung tissue as determined by IHC staining (light microscopy, × 200) and western blotting. Scale bars = 100 μm. The values are shown as the mean ± SD; = 6 mouse/group. ΨΨ P ## &&

机译：血清中咖啡因的浓度和肺中A R的表达。咖啡因治疗可显着增加血清中咖啡因的浓度，并降低肺中的A R蛋白水平。通过ELISA测定血清中咖啡因的浓度。通过IHC染色（光学显微镜，×200）和Western blotting检测，A R在肺组织中的表达。比例尺= 100μm。数值显示为平均值±SD。 = 6鼠/组。 P ## &&

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期刊名称 Respiratory Research
作者
Shangqin Chen; Qiuping Wu; Dingjuan Zhong; Changchong Li; Lizhong Du;
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作者单位

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年(卷),期 2020(21),-1
年度 2020
页码 -1
总页数 13
原文格式 PDF
正文语种
中图分类呼吸生理学 ;
关键词
Bronchopulmonary dysplasia; Oxidative stress; Caffeine; Inflammasome; NF-κB pathway;

机译：支气管肺发育不良;氧化应激;咖啡因;炎性体;NF-κB途径;

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专利

1. Caffeine prevents hyperoxia-induced lung injury in neonatal mice through NLRP3 inflammasome and NF-κB pathway [J] . Shangqin Chen, Qiuping Wu, Dingjuan Zhong, Respiratory Research . 2020 ,第1期

机译：咖啡因通过NLRP3炎症组和NF-κB途径预防新生儿小鼠中的高氧诱导的肺损伤
2. Apelin-13 Administration Protects Against LPS-Induced Acute Lung Injury by Inhibiting NF-κB Pathway and NLRP3 Inflammasome Activation [J] . Zhang H., Chen S., Zeng M., Cellular Physiology and Biochemistry . 2018 ,第5期

机译：Apelin-13给药可通过抑制NF-κB途径和NLRP3炎性体激活来保护LPS诱导的急性肺损伤。
3. Acetate Downregulates the Activation of NLRP3 Inflammasomes and Attenuates Lung Injury in Neonatal Mice With Bronchopulmonary Dysplasia [J] . Qian Zhang, Xiao Ran, Yu He, Frontiers in Pediatrics . 2020 ,第a期

机译：醋酸酯下调NLRP3炎性炎症的激活，并衰减用支气管扩张发育不良的新生儿小鼠的肺损伤
4. Neuroprotection of Glibenclamide against Brain Injury after Cardiac Arrest via Modulation of NLRP3 Inflammasome [C] . Xiuli Yang, Zhuoran Wang, Xiaofeng Jia Annual International Conference of the IEEE Engineering in Medicine and Biology Society . 2019

机译：格列本脲通过调节NLRP3炎症小体对心脏骤停后脑损伤的神经保护作用。
5. Thermodynamically-Constrained Computational Modeling of Lung Tissue Bioenergetics and the Effect of Hyperoxia-Induced Acute Lung Injury [D] . Zhang, Xiao. 2019

机译：热力学约束的肺组织生物能学计算模型及高氧血症引起的急性肺损伤的作用
6. Rhein Suppresses Lung Inflammatory Injury Induced by Human Respiratory Syncytial Virus Through Inhibiting NLRP3 Inflammasome Activation via NF-κB Pathway in Mice [O] . Cunsi Shen, Zhengguang Zhang, Tong Xie, 2019

机译：rhein通过抑制小鼠NF-κB途径抑制NLRP3炎症组血活化抑制人类呼吸合胞病毒诱导的肺炎损伤
7. Faculty Opinions recommendation of Suppression of NLRP3 inflammasome by erythropoietin via the EPOR/JAK2/STAT3 pathway contributes to attenuation of acute lung injury in mice. [O] . George Kasotakis, Brent Stanfield 2020

机译：教师意见通过Eythropoietin通过Eythropoietin抑制NLRP3炎症的推荐有助于抑制小鼠急性肺损伤。

Caffeine prevents hyperoxia-induced lung injury in neonatal mice through NLRP3 inflammasome and NF-κB pathway

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