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Novel Activating Mutations of the Calcium-Sensing Receptor: The Calcilytic NPS-2143 Mitigates Excessive Signal Transduction of Mutant Receptors

机译：钙敏感受体的新型激活突变：钙化NPS-2143减轻突变受体的过度信号转导。

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This article appears in The Journal of Clinical Endocrinology & Metabolism. 10.1210/jc.2010-0651

机译：本文发表在《临床内分泌与代谢杂志》上。 10.1210 / jc.2010-0651

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期刊名称 Molecular Endocrinology
作者
Saskia Letz; Ramona Rus; Christine Haag; Helmuth-Günther Dörr; Dirk Schnabel; Matthias Möhlig; Egbert Schulze; Karin Frank-Raue; Friedhelm Raue; Bernhard Mayr; Christof Schöfl;
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年(卷),期 -1(24),9
年度 -1
页码 1888
总页数 10
原文格式 PDF
正文语种
中图分类分子生物学;
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专利

1. Novel Activating Mutations of the Calcium-Sensing Receptor: The Calcilytic NPS-2143 Mitigates Excessive Signal Transduction of Mutant Receptors [J] . Saskia Letz, Ramona Rus, Christine Haag, The journal of clinical endocrinology and metabolism . 2010,第10期

机译：钙敏感受体的新型激活突变：钙化NPS-2143减轻突变受体的过度信号转导。
2. Activating mutations of the calcium-sensing receptor: The calcilytics ATF-936 and AXT-914 attenuate mutants causing autosomal dominant hypocalcemia and Bartter syndrome type 5 [J] . Experimental and clinical endocrinology and diabetes: Official journal, German Society of Endocrinology [and] German Diabetes Association . 2013,第3期

机译：钙敏感受体的激活突变：钙分解剂ATF-936和AXT-914减弱导致常染色体显性低血钙和5型Bartter综合征的突变体
3. Activating Calcium-Sensing Receptor Mutations: Prospects for Future Treatment with Calcilytics [J] . Mayr Bernhard, Glaudo Markus, Schoefl Christof Trends in Endocrinology and Metabolism: TEM . 2016,第9期

机译：激活钙敏感受体突变：钙盐抑制剂治疗的前景。
4. Cellular Automata Simulation of Signal Transduction Mediated by Healthy and Calcium-sensing Receptors [C] . YONGWIMON LENBURY, CHONTITA RATTANAKUL International Conference on Mathematical and Computational Methods in Science and Engineering . 2014

机译：通过健康和钙传感受体介导的信号转导的细胞自动机模拟
5. Activation of novel signal transduction pathways by human EP1 prostanoid receptors: The G-protein coupled receptors for prostaglandin E2. [D] . Ji, Ruyue. 2010

机译：人类EP1前列腺素受体激活新的信号转导途径：前列腺素E2的G蛋白偶联受体。
6. Amino Alcohol- (NPS-2143) and Quinazolinone-Derived Calcilytics (ATF936 and AXT914) Differentially Mitigate Excessive Signalling of Calcium-Sensing Receptor Mutants Causing Bartter Syndrome Type 5 and Autosomal Dominant Hypocalcemia [O] . Saskia Letz, Christine Haag, Egbert Schulze, -1

机译：氨基酒精（NPS-2143）和喹唑啉酮衍生的钙分解剂（ATF936和AXT914）差异性地减轻了钙敏感受体突变体的过度信号传递导致5型易感综合征和常染色体显性低血钙症。
7. Amino alcohol- (NPS-2143) and quinazolinone-derived calcilytics (ATF936 and AXT914) differentially mitigate excessive signalling of calcium-sensing receptor mutants causing Bartter syndrome Type 5 and autosomal dominant hypocalcemia. [O] . Saskia Letz, Christine Haag, Egbert Schulze, 2014

机译：氨基醇 - （Nps-2143）和喹唑啉酮衍生的钙质溶解剂（aTF936和aXT914）差异性地减轻钙敏感受体突变体的过度信号传导，导致5型Bartter综合征和常染色体显性低钙血症。

Novel Activating Mutations of the Calcium-Sensing Receptor: The Calcilytic NPS-2143 Mitigates Excessive Signal Transduction of Mutant Receptors

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