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SkM1 and Cx32 improve conduction in canine myocardial infarcts yet only SkM1 is antiarrhythmic

机译：SkM1和Cx32改善犬心肌梗死的传导但只有SkM1具有抗心律不齐的作用

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摘要

AimsReentry accounts for most life-threatening arrhythmias, complicating myocardial infarction, and therapies that consistently prevent reentry from occurring are lacking. In this study, we compare antiarrhythmic effects of gene transfer of green fluorescent protein (GFP; sham), the skeletal muscle sodium channel (SkM1), the liver-specific connexin (Cx32), and SkM1/Cx32 in the subacute canine infarct.

机译：AimsReentry可导致大多数危及生命的心律不齐，使心肌梗塞复杂化，并且缺乏能够始终阻止折返发生的疗法。在这项研究中，我们比较了亚急性犬梗死中绿色荧光蛋白（GFP; sham），骨骼肌钠通道（SkM1），肝脏特异性连接蛋白（Cx32）和SkM1 / Cx32基因转移的抗心律失常作用。

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期刊名称 Cardiovascular Research
作者
Gerard J.J. Boink; David H. Lau; Iryna N. Shlapakova; Eugene A. Sosunov; Evgeny P. Anyukhovsky; Helen E. Driessen; Wen Dun; Ming Chen; Peter Danilo Jr; Tove S. Rosen; Nazira Őzgen; Heather S. Duffy; Yelena Kryukova; Penelope A Boyden; Richard B. Robinson; Peter R. Brink; Ira S. Cohen; Michael R. Rosen;
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年(卷),期 -1(94),3
年度 -1
页码 450–459
总页数 10
原文格式 PDF
正文语种
中图分类心血管疾病;
关键词
Myocardial infarction Arrhythmias Nasup+/sup channels Connexins Gene therapy;

机译：心肌梗塞;心律不齐;Na sup + / sup通道;连接蛋白;基因治疗;

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专利

1. SkM1 and Cx32 improve conduction in canine myocardial infarcts yet only SkM1 is antiarrhythmic [J] . BoinkG.J.J., LauD.H., ShlapakovaI.N., Cardiovascular Research . 2012,第3期

机译：SkM1和Cx32改善犬心肌梗死的传导，但只有SkM1具有抗心律不齐的作用
2. SkM1 and Cx32 improve conduction in canine myocardial infarcts yet only SkM1 is antiarrhythmic [J] . Gerard J.J. Boink1234 David H. Lau1 Iryna N. Shlapakova15 Eugene A. Sosunov12 Evgeny P. Anyukhovsky12 Helen E. Driessen16 Wen Dun1 Ming Chen1 Peter Danilo Jr12 Tove S. Rosen7 Nazira Őzgen1 Heather S. Duffy18 Yelena Kryukova1 Penelope A Boyden12 Richard B. Robinson12 Peter R. Brink9 Ira S. Cohen9 and Michael R. Rosen1279* Cardiovascular Research . 2012,第3期

机译：SkM1和Cx32改善犬心肌梗死的传导，但只有SkM1具有抗心律不齐的作用
3. Total flavones from Elsholtzia blanda reduce infarct size and improve heart function during acute myocardial infarction by inhibiting myocardial apoptosis in canines. [J] . Ling H, Lou Y, Wu H, Acta Cardiologica . 2005,第3期

机译：急性Elsholtzia blanda的总黄酮可通过抑制犬的心肌细胞凋亡来减少梗塞面积并改善急性心肌梗塞期间的心脏功能。
4. Relation of Infarct Location and Size to Extent of Infarct Expansion After Acute Myocardial Infarction: A Quantitative Study Based on a Canine Model [C] . Jianhong Dou, Ling Xia, Yunliang Zang, International conference on life system modeling and simulation;International conference on intelligent computing for sustainable energy and environment;LSMS 2010;ICSEE 2010 . 2010

机译：急性心肌梗死后梗塞位置和大小与梗塞扩展程度的关系：基于犬模型的定量研究
5. Electrocardiographic imaging of paced sequences and infarct substrate in canine hearts, and in patients undergoing cardiac resynchronization therapy. [D] . Jia, Ping. 2004

机译：犬心脏以及接受心脏再同步治疗的患者中的起搏序列和梗塞底物的心电图成像。
6. Improved myocardial perfusion and cardiac function by controlled-release basic fibroblast growth factor using fibrin glue in a canine infarct model [O] . Shao-ping Nie, Xiao Wang, Shi-bin Qiao, 2010

机译：在犬梗死模型中使用纤维蛋白胶通过控释碱性成纤维细胞生长因子改善心肌灌注和心功能
7. Improved myocardial perfusion and cardiac function by controlled-release basic fibroblast growth factor using fibrin glue in a canine infarct model* [O] . Nie, Shao-ping, Wang, Xiao, Qiao, Shi-bin, 2010

机译：在犬梗死模型中使用纤维蛋白胶通过控释碱性成纤维细胞生长因子改善心肌灌注和心功能*
8. Protecting Ischemic Myocardium,Minimizing Infarct Size,and Enhancing Myocardial Repair. Myocardial Ischemia:Changes in Energy Conservation,and Specific Interventions to Improve Cardiac Contractility. [R] . jones,carl e. 1976

机译：保护缺血心肌，最大限度地减少梗塞面积，增强心肌修复能力。心肌缺血：能量保护的变化，以及改善心脏收缩力的特定干预措施。

SkM1 and Cx32 improve conduction in canine myocardial infarcts yet only SkM1 is antiarrhythmic

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