EFFECTS OF EXENDIN-4 ON ENDOPLASMIC RETICULUM STRESS-MEDIATED INSULIN RESISTANCE IN 3T3-L1 ADIPOCYTES

机译：Exendin-4对3T3-L1脂多糖内膜网状应力介导的胰岛素抵抗的影响

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ObjectiveEndoplasmic reticulum stress (ERS) is suspected as an important factor in the initiation of insulin resistance.

机译：目的怀疑内质网应激（ERS）是引发胰岛素抵抗的重要因素。

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期刊名称 Acta Endocrinologica (Bucharest)
作者
B. Guan; L. Wang; L. Ma; X. Liu; L. Liu;
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年(卷),期 2019(15),2
年度 2019
页码 158–164
总页数 7
原文格式 PDF
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Exendin-4 ER stress insulin resistance 3T3-L1 Adipocytes;

机译：Exendin-4;内质网应激;胰岛素抵抗;3T3-L1脂肪细胞;
入库时间 2022-08-21 10:49:31

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专利

1. Permethrin potentiates adipogenesis via intracellular calcium and endoplasmic reticulum stress-mediated mechanisms in 3T3-L1 adipocytes [J] . Xiao Xiao, Qi Weipeng, Clark John M., Food and Chemical Toxicology: An International Journal Published for the British Industrial Biological Research . 2017,第Pta1期

机译：氯蛋白通过细胞内钙和内质网胁迫介导机制在3T3-L1 adipocytes中提出脂肪生成
2. (-)-Hydroxycitric acid attenuates endoplasmic reticulum stress-mediated alterations in 3T3-L1 adipocytes by protecting mitochondria and downregulating inflammatory markers [J] . Nisha V. M., Priyanka A., Anusree S. S., Free radical research . 2014,第1a12期

机译：（-）-羟基柠檬酸通过保护线粒体和下调炎症标志物来减轻3T3-L1脂肪细胞内质网应激介导的变化
3. Phosphocreatine attenuates endoplasmic reticulum stress-mediated hepatocellular apoptosis ameliorates insulin resistance in diabetes model [J] . Jamalat Yazeed, Gamallat Yaser, Gislaine Pires Sanches Jaceline, Biochemical and Biophysical Research Communications . 2018,第3期

机译：磷酸官能衰减内质网胁迫介导的肝细胞凋亡改善糖尿病模型中的胰岛素抗性
4. MicroRNA Expression Profile in Insulin-Resistant 3T3-L1 Adipocyte [C] . H.Y. Ling, H.S. Ou, X.Y. Lei, World Congress on Heart Disease . 2007

机译：胰岛素抗性3T3-L1 adipocyte的MicroRNA表达谱
5. Potential role of endoplasmic reticulum redox changes in endoplasmic reticulum stress and impaired protein folding in obesity-associated insulin resistance [D] . Sarkar, Deboleena 2013

机译：内质网氧化还原变化在内质网应激和蛋白质折叠受损在肥胖相关胰岛素抵抗中的潜在作用
6. Prolonged insulin stimulation down-regulates GLUT4 through oxidative stress-mediated retromer inhibition by a protein kinase CK2-dependent mechanism in 3T3-L1 adipocytes. [O] . Jinhui Ma, Yuko Nakagawa, Itaru Kojima, 2014

机译：延长的胰岛素刺激通过蛋白激酶CK2依赖性机制在3T3-L1脂肪细胞中通过氧化应激介导的逆向异构体抑制而下调GLUT4。
7. Prolonged Insulin Stimulation Down-regulates GLUT4 through Oxidative Stress-mediated Retromer Inhibition by a Protein Kinase CK2-dependent Mechanism in 3T3-L1 Adipocytes [O] . Jinhui Ma, Yuko Nakagawa, Itaru Kojima, 2014

机译：延长胰岛素刺激通过氧化应激介导的回力抑制通过蛋白激酶CK2依赖性机制在3T3-L1 adipocytes中通过氧化应激介导的回力抑制来调节GLUT4

EFFECTS OF EXENDIN-4 ON ENDOPLASMIC RETICULUM STRESS-MEDIATED INSULIN RESISTANCE IN 3T3-L1 ADIPOCYTES

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