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Involvement of co-repressor LUH and the adapter proteins SLK1 and SLK2 in the regulation of abiotic stress response genes in Arabidopsis

机译:协同抑制因子LUH和衔接蛋白SLK1和SLK2参与拟南芥非生物胁迫响应基因的调控

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摘要

BackgroundDuring abiotic stress many genes that are important for growth and adaptation to stress are expressed at elevated levels. However, the mechanisms that keep the stress responsive genes from expressing under non stress conditions remain elusive. Recent genetic characterization of the co-repressor LEUNIG_HOMOLOG (LUH) and transcriptional adaptor proteins SEUSS-LIKE1 (SLK1) and SLK2 have been proposed to function redundantly in diverse developmental processes; however their function in the abiotic stress response is unknown. Moreover, the molecular functions of LUH, SLK1 and SLK2 remain obscure. Here, we show the molecular function of LUH, SLK1 and SLK2 and the role of this complex in the abiotic stress response.
机译:背景在非生物胁迫期间,许多对于生长和适应胁迫重要的基因均以升高的水平表达。但是,保持应激反应基因在非应激条件下表达的机制仍然难以捉摸。提出了共同抑制基因LEUNIG_HOMOLOG(LUH)和转录衔接蛋白SEUSS-LIKE1(SLK1)和SLK2的近期遗传学表征在多种发育过程中起着多余的作用。然而,它们在非生物应激反应中的功能尚不清楚。此外,LUH,SLK1和SLK2的分子功能仍然不清楚。在这里,我们显示了LUH,SLK1和SLK2的分子功能以及该复合物在非生物胁迫响应中的作用。

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