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Enrichment of IL‐17A+IFN‐γ+ and IL‐22+IFN‐γ+ T cell subsets is associated with reduction of NKp44+ILC3s in the terminal ileum of Crohns disease patients

机译：IL-17A +IFN-γ+和IL-22 +IFN-γ+ T细胞亚群的丰富与克罗恩病患者回肠末端NKp44 + ILC3s的减少有关

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期刊名称 Clinical and Experimental Immunology
作者
J. Li; A. L. Doty; Y. Tang; D. Berrie; A. Iqbal; S. A. Tan; M. J. Clare‐Salzler; S. M. Wallet; S. C. Glover;
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作者单位

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年(卷),期 2017(190),1
年度 2017
页码 143–153
总页数 11
原文格式 PDF
正文语种
中图分类免疫学;
关键词
Crohns disease innate lymphoid cells T cells;

机译：克罗恩病;先天淋巴样细胞;T细胞;

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专利

1. Enrichment of IL-17A(+)IFN-(+) and IL-22(+)IFN-(+) T cell subsets is associated with reduction of NKp44(+)ILC3s in the terminal ileum of Crohn's disease patients [J] . Li J., Doty A. L., Tang Y., Clinical and Experimental Immunology: An Official Journal of the British Society for Immunology . 2017,第1期

机译：IL-17A（+）IFN - （+）和IL-22（+）IFN - （+）T细胞亚群的富集与Crohn病人患者末尾的NKP44（+）ILC3S的减少有关
2. Effects of Anti-TNFα Treatment on Mucosal Expression of IL-17A, IL-21, and IL-22 and Cytokine-Producing T Cell Subsets in Crohn’s Disease [J] . Anders Dige, Maria K. Magnusson, Claus Uhrenholt, Mediators of inflammation . 2018,第6期

机译：抗TNFα处理对克罗恩病患者黏膜表达IL-17A，IL-21和IL-22以及产生细胞因子的T细胞亚群的影响
3. Effects of Anti-TNF alpha Treatment on Mucosal Expression of IL-17A, IL-21, and IL-22 and Cytokine-Producing T Cell Subsets in Crohn's Disease [J] . Dige Anders, Magnusson Maria K., Uhrenholt Claus, Mediators of inflammation . 2018,第Pta2期

机译：抗TNFα治疗对克罗恩病中IL-17A，IL-21和IL-22和细胞因子产生T细胞亚群的粘膜表达的影响
4. The Role of PSP (Policakarida Peptida) as Anti Inflammation Therapy and Vascular Endotel Cell Protectors in Dislipidemic Patients With or Without Diabetes Through IL1, TNF-α, CR CR, EPC, CEC In STEMI and NSTEMI Patients [C] . Djanggan Sargowo, Firyal Nadiah Rahmah, Fariz Wahyu Nugroho, International Conference on Bioinformatics and Nanomedicine from Natural Resources for Biomedical Research . 2019

机译：psp（policakarida peptida）作为抗炎患者的抗炎治疗和血管内核细胞保护剂在无异血患者中，通过IL1，TNF-α，CR CR，EPC，CEC，CEC，CEC在Stemi和NSTEMI患者中
5. Tumor-specific human primary T cells engineered to overexpress IL-2, IL-7, IL-15, or IL-21 confer varying degrees of tumor rejection and xenogeneic graft-versus-host disease in an immunodeficient mouse model. [D] . Markley, John Charles. 2009

机译：经过工程改造以过度表达IL-2，IL-7，IL-15或IL-21的肿瘤特异性人类原代T细胞可在免疫缺陷小鼠模型中赋予不同程度的肿瘤排斥反应和异种移植物抗宿主病。
6. Effects of Anti-TNFα Treatment on Mucosal Expression of IL-17A IL-21 and IL-22 and Cytokine-Producing T Cell Subsets in Crohns Disease [O] . Anders Dige, Maria K. Magnusson, Claus Uhrenholt, 2018

机译：抗TNFα处理对克罗恩病患者IL-17AIL-21和IL-22黏膜表达以及产生细胞因子的T细胞亚群的影响
7. P671 An IL23R risk variant is associated with changes in CD4+ T-cell subsets in peripheral blood of Crohn's disease patients [O] . T.B. Murdoch, A. Ueno, G. Kaplan, 2013

机译：P671 IL23R风险变体与CROHN病患者外周血中CD4 + T细胞亚群的变化相关

Enrichment of IL‐17A+IFN‐γ+ and IL‐22+IFN‐γ+ T cell subsets is associated with reduction of NKp44+ILC3s in the terminal ileum of Crohns disease patients

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